The Jackson toxic milk mouse as a model for copper loading

被引:51
作者
Coronado, V [1 ]
Nanji, M [1 ]
Cox, DW [1 ]
机构
[1] Univ Alberta, Dept Med Genet, Edmonton, AB T6G 2H7, Canada
关键词
D O I
10.1007/s00335-001-3021-y
中图分类号
Q5 [生物化学]; Q7 [分子生物学];
学科分类号
071010 ; 081704 ;
摘要
[No abstract available]
引用
收藏
页码:793 / 795
页数:3
相关论文
共 22 条
[1]   Characterization of copper interactions with Alzheimer amyloid β peptides:: Identification of an attomolar-affinity copper binding site on amyloid β1-42 [J].
Atwood, CS ;
Scarpa, RC ;
Huang, XD ;
Moir, RD ;
Jones, WD ;
Fairlie, DP ;
Tanzi, RE ;
Bush, AI .
JOURNAL OF NEUROCHEMISTRY, 2000, 75 (03) :1219-1233
[2]  
BIEMPICA L, 1988, LAB INVEST, V59, P500
[3]  
Bronson RT., 1995, MOUSE GENOME, V93, P152
[4]   Null mutation of the murine ATP7B (Wilson disease) gene results in intracellular copper accumulation and late-onset hepatic nodular transformation [J].
Buiakova, OI ;
Xu, J ;
Lutsenko, S ;
Zeitlin, S ;
Das, K ;
Das, S ;
Ross, BM ;
Mekios, C ;
Scheinberg, IH ;
Gilliam, TC .
HUMAN MOLECULAR GENETICS, 1999, 8 (09) :1665-1671
[5]   THE WILSON DISEASE GENE IS A PUTATIVE COPPER TRANSPORTING P-TYPE ATPASE SIMILAR TO THE MENKES GENE [J].
BULL, PC ;
THOMAS, GR ;
ROMMENS, JM ;
FORBES, JR ;
COX, DW .
NATURE GENETICS, 1993, 5 (04) :327-337
[6]  
Cookson MR, 1999, BRAIN PATHOL, V9, P165
[7]  
COX DW, 1998, SLEISENGER FORDTRANS, P1104
[8]  
Good PF, 1996, AM J PATHOL, V149, P21
[9]  
Ha-Hao Duc, 1998, European Journal of Human Genetics, V6, P616
[10]   OXYGEN-TOXICITY, OXYGEN RADICALS, TRANSITION-METALS AND DISEASE [J].
HALLIWELL, B ;
GUTTERIDGE, JMC .
BIOCHEMICAL JOURNAL, 1984, 219 (01) :1-14