Molecular mechanisms of amyloidosis

[1] INCREASED BODY-TEMPERATURE ACCELERATES AGGREGATION OF THE LEU-68 -] GLN MUTANT CYSTATIN-C, THE AMYLOID-FORMING PROTEIN IN HEREDITARY CYSTATIN-C AMYLOID ANGIOPATHY [J]. PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA, 1994, 91 (04) : 1416 - 1420

[2] Comparative studies of two transthyretin variants with protective effects on familial amyloidotic polyneuropathy: TTR R104H and TTR T119M [J]. BIOCHEMICAL AND BIOPHYSICAL RESEARCH COMMUNICATIONS, 2000, 270 (03) : 1024 - 1028

[3] Only amyloidogenic intermediates of transthyretin induce apoptosis [J]. BIOCHEMICAL AND BIOPHYSICAL RESEARCH COMMUNICATIONS, 2002, 294 (02) : 309 - 314

[4] Conformational switching and fibrillogenesis in the amyloidogenic fragment of apolipoprotein A-I [J]. JOURNAL OF BIOLOGICAL CHEMISTRY, 2003, 278 (04) : 2444 - 2451

[5] Light and electron microscopy immunohistochemical characterization of amyloid deposits [J]. AMYLOID-INTERNATIONAL JOURNAL OF EXPERIMENTAL AND CLINICAL INVESTIGATION, 1997, 4 (03): : 157 - 170

[6] Peripherally administered antibodies against amyloid β-peptide enter the central nervous system and reduce pathology in a mouse model of Alzheimer disease [J]. NATURE MEDICINE, 2000, 6 (08) : 916 - 919

[7] Review: Immunoglobulin light chain amyloidosis - The archetype of structural and pathogenic variability [J]. JOURNAL OF STRUCTURAL BIOLOGY, 2000, 130 (2-3) : 280 - 289

[8] A new human hereditary amyloidosis: The result of a stop-codon mutation in the apolipoprotein AII gene [J]. GENOMICS, 2001, 72 (03) : 272 - 277

[9] Booth D R, 2000, Hum Mutat, V16, P180, DOI 10.1002/1098-1004(200008)16:2<180::AID-HUMU20>3.0.CO

[10] 2-#

[1] INCREASED BODY-TEMPERATURE ACCELERATES AGGREGATION OF THE LEU-68 -] GLN MUTANT CYSTATIN-C, THE AMYLOID-FORMING PROTEIN IN HEREDITARY CYSTATIN-C AMYLOID ANGIOPATHY [J]. PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA, 1994, 91 (04) : 1416 - 1420

[2] Comparative studies of two transthyretin variants with protective effects on familial amyloidotic polyneuropathy: TTR R104H and TTR T119M [J]. BIOCHEMICAL AND BIOPHYSICAL RESEARCH COMMUNICATIONS, 2000, 270 (03) : 1024 - 1028

[3] Only amyloidogenic intermediates of transthyretin induce apoptosis [J]. BIOCHEMICAL AND BIOPHYSICAL RESEARCH COMMUNICATIONS, 2002, 294 (02) : 309 - 314

[4] Conformational switching and fibrillogenesis in the amyloidogenic fragment of apolipoprotein A-I [J]. JOURNAL OF BIOLOGICAL CHEMISTRY, 2003, 278 (04) : 2444 - 2451

[5] Light and electron microscopy immunohistochemical characterization of amyloid deposits [J]. AMYLOID-INTERNATIONAL JOURNAL OF EXPERIMENTAL AND CLINICAL INVESTIGATION, 1997, 4 (03): : 157 - 170

[6] Peripherally administered antibodies against amyloid β-peptide enter the central nervous system and reduce pathology in a mouse model of Alzheimer disease [J]. NATURE MEDICINE, 2000, 6 (08) : 916 - 919

[7] Review: Immunoglobulin light chain amyloidosis - The archetype of structural and pathogenic variability [J]. JOURNAL OF STRUCTURAL BIOLOGY, 2000, 130 (2-3) : 280 - 289

[8] A new human hereditary amyloidosis: The result of a stop-codon mutation in the apolipoprotein AII gene [J]. GENOMICS, 2001, 72 (03) : 272 - 277

[9] Booth D R, 2000, Hum Mutat, V16, P180, DOI 10.1002/1098-1004(200008)16:2<180::AID-HUMU20>3.0.CO

[10] 2-#