Cure of apparent mineralocorticoid excess by kidney transplantation

被引:42
作者
Palermo, M [1 ]
Cossu, M
Shackleton, CHL
机构
[1] Univ Sassari, I-07100 Sassari, Italy
[2] SS Annunziata Hosp, Sassari, Italy
[3] Childrens Hosp Oakland, Res Inst, Oakland, CA 94609 USA
关键词
D O I
10.1056/NEJM199812103392414
中图分类号
R5 [内科学];
学科分类号
1002 ; 100201 ;
摘要
引用
收藏
页码:1787 / 1788
页数:2
相关论文
共 4 条
[1]   Molecular basis for hypertension in the "type II variant" of apparent mineralocorticoid excess [J].
Li, A ;
Tedde, R ;
Krozowski, ZS ;
Pala, A ;
Li, KXZ ;
Shackleton, CHL ;
Mantero, F ;
Palermo, M ;
Stewart, PM .
AMERICAN JOURNAL OF HUMAN GENETICS, 1998, 63 (02) :370-379
[2]   Apparent mineralocorticoid excess: Genotype is correlated with biochemical phenotype [J].
Mune, T ;
White, PC .
HYPERTENSION, 1996, 27 (06) :1193-1199
[3]   Urinary free cortisone and the assessment of 11 beta-hydroxysteroid dehydrogenase activity in man [J].
Palermo, M ;
Shackleton, CHL ;
Mantero, F ;
Stewart, PM .
CLINICAL ENDOCRINOLOGY, 1996, 45 (05) :605-611
[4]   A genetic defect resulting in mild low-renin hypertension [J].
Wilson, RC ;
Dave-Sharma, S ;
Wei, JQ ;
Obeyesekere, VR ;
Li, K ;
Ferrari, P ;
Krozowski, ZS ;
Shackleton, CHL ;
Bradlow, L ;
Wiens, T ;
New, MI .
PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA, 1998, 95 (17) :10200-10205