Ion channels - A new wave for heart rhythms

[1] THE PROTEIN ISK IS A DUAL ACTIVATOR OF K+ AND CL- CHANNELS [J]. NATURE, 1993, 365 (6449) : 850 - 852

[2] K(v)LQT1 and IsK (minK) proteins associate to form the I-Ks cardiac potassium current [J]. NATURE, 1996, 384 (6604) : 78 - 80

[3] BUSCH AE, 1994, MOL PHARMACOL, V46, P750

[4] A MOLECULAR-BASIS FOR CARDIAC-ARRHYTHMIA - HERG MUTATIONS CAUSE LONG QT SYNDROME [J]. CELL, 1995, 80 (05) : 795 - 803

[5] SITE-SPECIFIC MUTATIONS IN A MINIMAL VOLTAGE-DEPENDENT K+ CHANNEL ALTER ION SELECTIVITY AND OPEN-CHANNEL BLOCK [J]. NEURON, 1991, 7 (03) : 403 - 408

[6] Coassembly of K(v)LQT1 and minK (IsK) proteins to form cardiac I-Ks potassium channel [J]. NATURE, 1996, 384 (6604) : 80 - 83

[7] CLONING OF A MEMBRANE-PROTEIN THAT INDUCES A SLOW VOLTAGE-GATED POTASSIUM CURRENT [J]. SCIENCE, 1988, 242 (4881) : 1042 - 1045

[8] Positional cloning of a novel potassium channel gene: KVLQT1 mutations cause cardiac arrhythmias [J]. NATURE GENETICS, 1996, 12 (01) : 17 - 23

[1] THE PROTEIN ISK IS A DUAL ACTIVATOR OF K+ AND CL- CHANNELS [J]. NATURE, 1993, 365 (6449) : 850 - 852

[2] K(v)LQT1 and IsK (minK) proteins associate to form the I-Ks cardiac potassium current [J]. NATURE, 1996, 384 (6604) : 78 - 80

[3] BUSCH AE, 1994, MOL PHARMACOL, V46, P750

[4] A MOLECULAR-BASIS FOR CARDIAC-ARRHYTHMIA - HERG MUTATIONS CAUSE LONG QT SYNDROME [J]. CELL, 1995, 80 (05) : 795 - 803

[5] SITE-SPECIFIC MUTATIONS IN A MINIMAL VOLTAGE-DEPENDENT K+ CHANNEL ALTER ION SELECTIVITY AND OPEN-CHANNEL BLOCK [J]. NEURON, 1991, 7 (03) : 403 - 408

[6] Coassembly of K(v)LQT1 and minK (IsK) proteins to form cardiac I-Ks potassium channel [J]. NATURE, 1996, 384 (6604) : 80 - 83

[7] CLONING OF A MEMBRANE-PROTEIN THAT INDUCES A SLOW VOLTAGE-GATED POTASSIUM CURRENT [J]. SCIENCE, 1988, 242 (4881) : 1042 - 1045

[8] Positional cloning of a novel potassium channel gene: KVLQT1 mutations cause cardiac arrhythmias [J]. NATURE GENETICS, 1996, 12 (01) : 17 - 23