Inhibition of NUDEL (nuclear distribution element-like)-oligopeptidase activity by disrupted-in-schizophrenia 1

被引:61
作者
Hayashi, MAF
Portaro, FCV
Bastos, MF
Guerreiro, JR
Oliveira, V
Gorrao, SS
Tambourgi, DV
Sant'Anna, OA
Whiting, PJ
Camargo, LM
Konno, K
Brandon, NJ
Camargo, ACM
机构
[1] Merck Sharp & Dohme Ltd, Harlow CM20 2QR, Essex, England
[2] Univ Cidade Sao Paulo, CAT, CEPID, BR-05503900 Sao Paulo, Brazil
[3] Univ Cidade Sao Paulo, Butantan Inst, Immunogenet Lab, BR-05503900 Sao Paulo, Brazil
[4] Univ Cidade Sao Paulo, Butantan Inst, Lab Immunochem, BR-05503900 Sao Paulo, Brazil
[5] Univ Cidade Sao Paulo, Neurosci Lab, BR-05503900 Sao Paulo, Brazil
关键词
endooligopeptidase A; neuropeptide metabolism; cysteine peptidase; neurodevelopmental pathology;
D O I
10.1073/pnas.0500330102
中图分类号
O [数理科学和化学]; P [天文学、地球科学]; Q [生物科学]; N [自然科学总论];
学科分类号
07 ; 0710 ; 09 ;
摘要
Recently, nuclear distribution element-like (NUDEL) has been implicated to play a role in lissencephaly and schizophrenia through interactions with the lissencephaly gene 1 (Lis1) and disrupted-in-schizophrenia 1 (DISC1) products, respectively. Interestingly, NUDEL is the same protein as endooligopeptidase A (EOPA), a thiol-activated peptidase involved in conversion and inactivation of a number of bioactive peptides. In this study, we have cloned EOPA from the human brain and have confirmed that it is equivalent to NUDEL, leading us to suggest a single name, NUDEL-oligopeptidase. In the brain, the monomeric form of NUDEL-oligopeptidase is responsible for the peptidase activity whose catalytic mechanism is likely to involve a reactive cysteine, because mutation of Cys-273 fully abolished NUDEL-oligopeptidase activity without disrupting the protein's secondary structure. Cys-273 is very close to the DISC1-binding site on NUDEL-oligopeptidase. Intriguingly, DISC1 inhibits NUDEL-oligopeptidase activity in a competitive fashion. We suggest that the activity of NUDEL-oligopeptidase is under tight regulation through protein-protein interactions and that disruption of these interactions, as postulated in a Scottish DISC1 translocation schizophrenia cohort, may lead to aberrant regulation of NUDEL-oligopeptidase, perhaps providing a substrate for the pathology of schizophrenia.
引用
收藏
页码:3828 / 3833
页数:6
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