Epigenetic Therapy for Breast Cancer

被引:33
作者
Cai, Feng-Feng [1 ]
Kohler, Corina [1 ]
Zhang, Bei [1 ]
Wang, Ming-Hong [2 ]
Chen, Wei-Jie [1 ]
Zhong, Xiao-Yan [1 ]
机构
[1] Univ Basel, Womens Hosp, Dept Biomed, Lab Gynecol Oncol, CH-4031 Basel, Switzerland
[2] Southeast Univ, Zhongda Hosp, Dept Gen Practice Med, Nanjing 210009, Jiangsu, Peoples R China
关键词
breast cancer; epigenetic therapy; DNA methylation inhibitors; Histone deacetylation inhibitors; HISTONE DEACETYLASE INHIBITOR; TUMOR-SUPPRESSOR GENES; DNA METHYLTRANSFERASE; IN-VIVO; PHASE-I; CYCLIC TETRAPEPTIDE; TRANSFORMED-CELLS; TEA POLYPHENOLS; ER-ALPHA; METHYLATION;
D O I
10.3390/ijms12074465
中图分类号
Q5 [生物化学]; Q7 [分子生物学];
学科分类号
070307 [化学生物学]; 071010 [生物化学与分子生物学];
摘要
Both genetic and epigenetic alterations can control the progression of cancer. Genetic alterations are impossible to reverse, while epigenetic alterations are reversible. This advantage suggests that epigenetic modifications should be preferred in therapy applications. DNA methyltransferases and histone deacetylases have become the primary targets for studies in epigenetic therapy. Some DNA methylation inhibitors and histone deacetylation inhibitors are approved by the US Food and Drug Administration as anti-cancer drugs. Therefore, the uses of epigenetic targets are believed to have great potential as a lasting favorable approach in treating breast cancer.
引用
收藏
页码:4465 / 4476
页数:12
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