Endoplasmic reticulum stress signalling - from basic mechanisms to clinical applications

被引:925
作者
Almanza, Aitor [1 ]
Carlesso, Antonio [2 ]
Chintha, Chetan [1 ]
Creedican, Stuart [3 ]
Doultsinos, Dimitrios [4 ,5 ]
Leuzzi, Brian [1 ]
Luis, Andreia [6 ]
McCarthy, Nicole [7 ]
Montibeller, Luigi [8 ]
More, Sanket [9 ]
Papaioannou, Alexandra [4 ,5 ]
Pueschel, Franziska [10 ]
Sassano, Maria Livia [9 ]
Skoko, Josip [11 ]
Agostinis, Patrizia [9 ]
de Belleroche, Jackie [8 ]
Eriksson, Leif A. [2 ]
Fulda, Simone [7 ]
Gorman, Adrienne M. [1 ]
Healy, Sandra [1 ]
Kozlov, Andrey [6 ]
Munoz-Pinedo, Cristina [10 ]
Rehm, Markus [11 ]
Chevet, Eric [4 ,5 ]
Samali, Afshin [1 ]
机构
[1] Natl Univ Ireland, Apoptosis Res Ctr, Galway, Ireland
[2] Univ Gothenburg, Dept Chem & Mol Biol, Gothenburg, Sweden
[3] Randox Teoranta, Dungloe, Cty Donegal, Ireland
[4] Univ Rennes, INSERM U1242, Rennes, France
[5] Ctr Lutte Canc Eugene Marquis, Rennes, France
[6] AUVA Res Ctr, Ludwig Boltzmann Inst Expt & Clin Traumatol, Vienna, Austria
[7] Goethe Univ, Inst Expt Canc Res Paediat, Frankfurt, Germany
[8] Imperial Coll London, Fac Med, Div Brain Sci, Neurogenet Grp, London, England
[9] Katholieke Univ Leuven, Dept Cellular & Mol Med, Lab Cell Death & Therapy, Leuven, Belgium
[10] Bellvitge Biomed Res Inst IDIBELL, Oncobell Program, Cell Death Regulat Grp, Barcelona, Spain
[11] Univ Stuttgart, Inst Cell Biol & Immunol, Stuttgart, Germany
基金
欧盟地平线“2020”;
关键词
endoplasmic reticulum; proteostasis; signalling pathway; stress; UNFOLDED PROTEIN RESPONSE; TO-MESENCHYMAL TRANSITION; PLASMA-CELL DIFFERENTIATION; PANCREATIC BETA-CELLS; BOX-BINDING PROTEIN-1; MESSENGER-RNA DECAY; ER-STRESS; TRANSCRIPTION FACTOR; QUALITY-CONTROL; GLUCOSE-HOMEOSTASIS;
D O I
10.1111/febs.14608
中图分类号
Q5 [生物化学]; Q7 [分子生物学];
学科分类号
070307 [化学生物学]; 071010 [生物化学与分子生物学];
摘要
The endoplasmic reticulum (ER) is a membranous intracellular organelle and the first compartment of the secretory pathway. As such, the ER contributes to the production and folding of approximately one-third of cellular proteins, and is thus inextricably linked to the maintenance of cellular homeostasis and the fine balance between health and disease. Specific ER stress signalling pathways, collectively known as the unfolded protein response (UPR), are required for maintaining ER homeostasis. The UPR is triggered when ER protein folding capacity is overwhelmed by cellular demand and the UPR initially aims to restore ER homeostasis and normal cellular functions. However, if this fails, then the UPR triggers cell death. In this review, we provide a UPR signalling-centric view of ER functions, from the ER's discovery to the latest advancements in the understanding of ER and UPR biology. Our review provides a synthesis of intracellular ER signalling revolving around proteostasis and the UPR, its impact on other organelles and cellular behaviour, its multifaceted and dynamic response to stress and its role in physiology, before finally exploring the potential exploitation of this knowledge to tackle unresolved biological questions and address unmet biomedical needs. Thus, we provide an integrated and global view of existing literature on ER signalling pathways and their use for therapeutic purposes.
引用
收藏
页码:241 / 278
页数:38
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