共 66 条
[1]
Blockade of T lymphocyte costimulation with cytotoxic T lymphocyte-associated antigen 4-immunoglobulin (CTLA4Ig) reverses the cellular pathology of psoriatic plaques, including the activation of keratinocytes, dendritic cells, and endothelial cells
[J].
JOURNAL OF EXPERIMENTAL MEDICINE,
2000, 192 (05)
:681-693
[4]
Austin L, 1998, J INVEST DERMATOL, V110, P649
[6]
The majority of epidermal T cells in Psoriasis vulgaris lesions can produce type 1 cytokines, interferon-γ, interleukin-2, and tumor necrosis factor-α, defining TC1 (cytotoxic T lymphocyte) and TH1 effector populations:: a type 1 differentiation bias is also measured in circulating blood T cells in psoriatic patients
[J].
JOURNAL OF INVESTIGATIVE DERMATOLOGY,
1999, 113 (05)
:752-759
[7]
BAKER BS, 1985, CLIN EXP IMMUNOL, V61, P526
[9]
BAKER BS, 1984, BRIT J DERMATOL, V110, P555