The clinical and diagnostic implications of mosaicism in the neurofibromatoses

被引:233
作者
Ruggieri, M
Huson, SM
机构
[1] CNR, IBFSNC, Inst Bioimaging & Pathophysiol Cent Nervous Syst, I-95125 Catania, Italy
[2] Univ Catania, Dept Pediat, I-95124 Catania, Italy
[3] Oxford Radcliffe Hosp, Dept Clin Genet, Oxford, England
关键词
D O I
10.1212/WNL.56.11.1433
中图分类号
R74 [神经病学与精神病学];
学科分类号
摘要
Neurofibromatosis type 1 and type 2 both occur in mosaic forms. Mosaicism results from somatic mutations. Early somatic mutations cause generalized disease, clinically indistinguishable from nonmosaic forms. Later somatic mutation gives rise to localized disease often described as segmental. In individuals with mosaic or localized manifestations of neurofibromatosis type 1 (segmental neurofibromatosis type 1), disease features are limited to the affected area, which varies from a narrow strip to one quadrant and occasionally to one half of the body. Distribution is usually unilateral but can be bilateral, either in a symmetric or asymmetrical arrangement. Patients with localized neurofibromatosis type 2 have disease-related tumors localized to one part of the nervous system; for example a unilateral vestibular schwannoma with ipsilateral meningiomas or multiple schwannomas in one part of the peripheral nervous system. The recognition of mosaic phenotypes is important. Individuals with the mosaic form, even with a generalized phenotype, are less likely to have severe disease. They also have lower offspring recurrence risk than individuals with the nonmosaic form. The mosaic forms of neurofibromatosis provide a good example of the effects of somatic mutation. It is increasingly recognized that mild and unusual forms of many dominantly inherited disorders are caused by the same mechanism.
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页码:1433 / 1443
页数:11
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