Enhanced external counterpulsation: Mechanism of action

被引：20

作者：

Feldman, AM ^{[1
]}

机构：

[1] Univ Pittsburgh, Div Cardiol, Pittsburgh, PA USA

来源：

CLINICAL CARDIOLOGY | 2002年 / 25卷 / 12期

关键词：

angiogenic growth factors; atrial natriuretic peptide; brain natriuretic peptide; endothelian; enhanced external counterpulsation; MCP-1; nitric oxide; vascular endothelial growth factor;

D O I：

10.1002/clc.4960251405

中图分类号：

R5 [内科学];

学科分类号：

1002 [临床医学]; 100201 [内科学];

摘要：

The mechanisms accounting for the salutary benefits of enhanced external counterpulsation (EECP) remain largely unknown. Emerging data now point to three possible hypotheses to explain its efficacy. These possible explanations include enhanced diastolic flow, changes in the neurohumoral milieu, and changes in ventricular function independent of changes in cardiac load. While each of these three hypotheses may help explain the mechanism behind EECP's effects, more clinical studies are required to refine these proposed explanations.

引用

页码：11 / 15

页数：5

共 12 条

[1]

Gorcsan J, 2000, J AM COLL CARDIOL, V35, p230A

[2]

A NEW THERAPEUTIC APPROACH TO ACUTE CORONARY OCCLUSION .2. OPENING DORMANT CORONARY COLLATERAL CHANNELS BY COUNTERPULSATION [J].