The chronic colitis developed by HLA-B27 transgenic rats is associated with altered in vivo mucin synthesis

被引:24
作者
Faure, M
Moënnoz, D
Mettraux, C
Montigon, F
Schiffrin, EJ
Obled, C
Breuillé, D
Boza, J
机构
[1] Nestec Ltd, Nestle Res Ctr, Nutr & Hlth Dept, CH-1000 Lausanne 26, Switzerland
[2] INRA, Ctr Clermont Ferrand Theix, Unite Nutr & Metab Prot, F-63122 Ceyrat, France
关键词
mucin; synthesis; chronic colitis; IBD; HLA-B27; rats;
D O I
10.1023/B:DDAS.0000017462.75257.70
中图分类号
R57 [消化系及腹部疾病];
学科分类号
摘要
HLA-B27 transgenic rats spontaneously developing a chronic inflammation mainly involving the colon are recognized as a powerful animal model for IBD. We investigated the mucin production in 6-month-old HLA-B27 rats by measuring in vivo fractional synthesis rate (FSR) and expression of mucins. In the inflamed colon of HLA-B27 rats, the mucin FSR was stimulated by 75% compared to F-344 controls, while MUC2,3 mRNA expression was unchanged. A local depletion in mucus-containing goblet cells was observed, suggesting a rapid mucin production/release and/or a real global decrease in goblet cell number. In the noninflamed jejunum of HLA-B27 rats, the mucin FSR was reduced by 35% compared to controls, while MUC2,3 mRNA expression was unchanged. Different alterations in mucin metabolism and expression are observed between HLA-B27 rats and a model of chemically induced chronic colitis (DSS-treated rats), suggesting that mucin alterations may be dependent on the animal model and colitis underlying mechanism.
引用
收藏
页码:339 / 346
页数:8
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