p53, the cellular gatekeeper for growth and division

被引:6535
作者
Levine, AJ
机构
[1] Department of Molecular Biology, Lewis Thomas Laboratory, Princeton University, Princeton
关键词
D O I
10.1016/S0092-8674(00)81871-1
中图分类号
Q5 [生物化学]; Q7 [分子生物学];
学科分类号
071010 ; 081704 ;
摘要
[No abstract available]
引用
收藏
页码:323 / 331
页数:9
相关论文
共 55 条
[1]   Specific P53 mutations are associated with de novo resistance to doxorubicin in breast cancer patients [J].
Aas, T ;
Borresen, AL ;
Geisler, S ;
SmithSorensen, B ;
Johnsen, H ;
Varhaug, JE ;
Akslen, LA ;
Lonning, PE .
NATURE MEDICINE, 1996, 2 (07) :811-814
[2]  
BOND JA, 1995, CANCER RES, V55, P2404
[3]   BOTH VIRAL (ADENOVIRUS E1B) AND CELLULAR (HSP-70, P53) COMPONENTS INTERACT WITH CENTROSOMES [J].
BROWN, CR ;
DOXSEY, SJ ;
WHITE, E ;
WELCH, WJ .
JOURNAL OF CELLULAR PHYSIOLOGY, 1994, 160 (01) :47-60
[4]   INDUCTION OF THE GROWTH INHIBITOR IGF-BINDING PROTEIN-3 BY P53 [J].
BUCKBINDER, L ;
TALBOTT, R ;
VELASCOMIGUEL, S ;
TAKENAKA, I ;
FAHA, B ;
SEIZINGER, BR ;
KLEY, N .
NATURE, 1995, 377 (6550) :646-649
[5]   P53-DEPENDENT APOPTOSIS IN THE ABSENCE OF TRANSCRIPTIONAL ACTIVATION OF P53-TARGET GENES [J].
CAELLES, C ;
HELMBERG, A ;
KARIN, M .
NATURE, 1994, 370 (6486) :220-223
[6]   CRYSTAL-STRUCTURE OF A P53 TUMOR-SUPPRESSOR DNA COMPLEX - UNDERSTANDING TUMORIGENIC MUTATIONS [J].
CHO, YJ ;
GORINA, S ;
JEFFREY, PD ;
PAVLETICH, NP .
SCIENCE, 1994, 265 (5170) :346-355
[7]   A P53-DEPENDENT MOUSE SPINDLE CHECKPOINT [J].
CROSS, SM ;
SANCHEZ, CA ;
MORGAN, CA ;
SCHIMKE, MK ;
RAMEL, S ;
IDZERDA, RL ;
RASKIND, WH ;
REID, BJ .
SCIENCE, 1995, 267 (5202) :1353-1356
[8]   WILD-TYPE P53 MEDIATES APOPTOSIS BY E1A, WHICH IS INHIBITED BY E1B [J].
DEBBAS, M ;
WHITE, E .
GENES & DEVELOPMENT, 1993, 7 (04) :546-554
[9]  
DELSAL G, 1995, MOL CELL BIOL, V15, P7152
[10]   MICE LACKING P21(C/P1/WAF1) UNDERGO NORMAL DEVELOPMENT, BUT ARE DEFECTIVE IN G1 CHECKPOINT CONTROL [J].
DENG, CX ;
ZHANG, PM ;
HARPER, JW ;
ELLEDGE, SJ ;
LEDER, P .
CELL, 1995, 82 (04) :675-684