Heart failure - A state of brain natriuretic peptide deficiency or resistance or both!

被引：60

作者：

Chen, Horng H. ^{[1
]}

机构：

[1] Mayo Clin, Coll Med, Dept Internal Med, Div Cardiovasc Dis, Rochester, MN USA

来源：

JOURNAL OF THE AMERICAN COLLEGE OF CARDIOLOGY | 2007年 / 49卷 / 10期

关键词：

D O I：

10.1016/j.jacc.2006.12.013

中图分类号：

R5 [内科学];

学科分类号：

1002 ; 100201 ;

摘要：

引用

页码：1089 / 1091

页数：3

共 17 条

[1] Mechanisms of renal hyporesponsiveness to ANP in heart failure [J].

Charloux, A ;

Piquard, F ;

Doutreleau, S ;

Brandenberger, G ;

Geny, B .

EUROPEAN JOURNAL OF CLINICAL INVESTIGATION, 2003, 33 (09) :769-778

[2] Renal response to acute neutral endopeptidase inhibition in mild and severe experimental heart failure [J].

Chen, HH ;

Schirger, JA ;

Chau, WL ;

Jougasaki, M ;

Lisy, O ;

Redfield, MM ;

Barclay, PT ;

Burnett, JC .

CIRCULATION, 1999, 100 (24) :2443-2448

[3] The natriuretic peptides in heart failure: Diagnostic and therapeutic potentials [J].

Chen, HH ;

Burnett, JC .

PROCEEDINGS OF THE ASSOCIATION OF AMERICAN PHYSICIANS, 1999, 111 (05) :406-416

[4] Maximizing the renal cyclic 3′-5′-guanosine monophosphate system with type V phosphodiesterase inhibition and exogenous natriuretic peptide:: A novel strategy to improve renal function in experimental overt heart failure [J].

Chen, Horng H. ;

Huntley, Brenda K. ;

Schirger, John A. ;

Cataliotti, Alessandro ;

Burnett, John C., Jr. .

JOURNAL OF THE AMERICAN SOCIETY OF NEPHROLOGY, 2006, 17 (10) :2742-2747

[5] Acute phosphodiesterase 5 inhibition mimics hemodynamic effects of B-type natriuretic peptide and potentiates B-type natriuretic peptide effects in failing but not normal canine heart [J].

Forfia, Paul R. ;

Lee, Myung ;

Tunin, Richard S. ;

Mahmud, Mobusher ;

Champion, Hunter C. ;

Kass, David A. .

JOURNAL OF THE AMERICAN COLLEGE OF CARDIOLOGY, 2007, 49 (10) :1079-1088

[6] COMPARISON OF NEUROENDOCRINE ACTIVATION IN PATIENTS WITH LEFT-VENTRICULAR DYSFUNCTION WITH AND WITHOUT CONGESTIVE-HEART-FAILURE - A SUBSTUDY OF THE STUDIES OF LEFT-VENTRICULAR DYSFUNCTION (SOLVD) [J].

FRANCIS, GS ;

BENEDICT, C ;

JOHNSTONE, DE ;

KIRLIN, PC ;

NICKLAS, J ;

LIANG, CS ;

KUBO, SH ;

RUDINTORETSKY, E ;

YUSUF, S .

CIRCULATION, 1990, 82 (05) :1724-1729

[7] Quantitative mass spectral evidence for the absence of circulating brain natriuretic peptide (BNP-32) in severe human heart failure [J].

Hawkridge, AM ;

Heublein, DM ;

Bergen, HR ;

Cataliotti, A ;

Burnett, JC ;

Muddiman, DC .

PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA, 2005, 102 (48) :17442-17447

[8] Evidence for functional heterogeneity of circulating B-type natriuretic peptide [J].

Liang, Faquan ;

O'Rear, Jessica ;

Schellenberger, Ute ;

Tai, Lungkuo ;

Lasecki, Michael ;

Schreiner, George F. ;

Apple, Fred S. ;

Maisel, Alan S. ;

Pollitt, N. Stephen ;

Protter, Andrew A. .

JOURNAL OF THE AMERICAN COLLEGE OF CARDIOLOGY, 2007, 49 (10) :1071-1078

[9] Differential atrial and ventricular expression of myocardial BNP during evolution of heart failure [J].

Luchner, A ;

Stevens, TL ;

Borgeson, DD ;

Redfield, M ;

Wei, CM ;

Porter, JG ;

Burnett, JC .

AMERICAN JOURNAL OF PHYSIOLOGY-HEART AND CIRCULATORY PHYSIOLOGY, 1998, 274 (05) :H1684-H1689

[10] FAILURE OF ATRIAL NATRIURETIC FACTOR TO INCREASE WITH VOLUME EXPANSION IN ACUTE AND CHRONIC CONGESTIVE HEART-FAILURE IN THE DOG [J].

REDFIELD, MM ;

EDWARDS, BS ;

MCGOON, MD ;

HEUBLEIN, DM ;

AARHUS, LL ;

BURNETT, JC .

CIRCULATION, 1989, 80 (03) :651-657

← 1 2 →