Tumor necrosis factor-α, sphingosine, ceramide:: Which is the appropriate marker of inflammation?

被引：5

作者：

Horton, JW ^{[1
]}

机构：

[1] SW Texas State Univ, Ctr Med, Dallas, TX 78666 USA

来源：

CRITICAL CARE MEDICINE | 1999年 / 27卷 / 11期

关键词：

sphingomyelin; ceramide; cytokines; tumor necrosis factor-alpha; multiple organ dysfunction;

D O I：

10.1097/00003246-199911000-00048

中图分类号：

R4 [临床医学];

学科分类号：

1002 ; 100602 ;

摘要：

引用

页码：2580 / 2581

页数：2

共 21 条

[1] EFFICACY AND SAFETY OF MONOCLONAL-ANTIBODY TO HUMAN TUMOR-NECROSIS-FACTOR-ALPHA IN PATIENTS WITH SEPSIS SYNDROME - A RANDOMIZED, CONTROLLED, DOUBLE-BLIND, MULTICENTER CLINICAL-TRIAL [J].

ABRAHAM, E ;

WUNDERINK, R ;

SILVERMAN, H ;

PERL, TM ;

NASRAWAY, S ;

LEVY, H ;

BONE, R ;

WENZEL, RP ;

BALK, R ;

ALLRED, R ;

PENNINGTON, JE ;

WHERRY, JC .

JAMA-JOURNAL OF THE AMERICAN MEDICAL ASSOCIATION, 1995, 273 (12) :934-941

[2] SEPSIS THERAPY TRIALS - CONTINUED DISAPPOINTMENT OR REASON FOR HOPE [J].

ABRAHAM, E ;

RAFFIN, TA .

JAMA-JOURNAL OF THE AMERICAN MEDICAL ASSOCIATION, 1994, 271 (23) :1876-1878

[3] Tumor necrosis factor-α and interleukin-1β synergistically depress human myocardial function [J].

Cain, BS ;

Meldrum, DR ;

Dinarello, CA ;

Meng, XZ ;

Joo, KS ;

Banerjee, A ;

Harken, AH .

CRITICAL CARE MEDICINE, 1999, 27 (07) :1309-1318

[4] Tumor necrosis factor mediates impaired wound healing in chronic abdominal sepsis [J].

Cooney, R ;

Iocono, J ;

Maish, G ;

Smith, JS ;

Ehrlich, P .

JOURNAL OF TRAUMA-INJURY INFECTION AND CRITICAL CARE, 1997, 42 (03) :415-420

[5] Ceramide concentrations in septic patients: A possible marker of multiple organ dysfunction syndrome [J].

Delogu, G ;

Famularo, G ;

Amati, F ;

Signore, L ;

Antonucci, A ;

Trinchieri, V ;

Di Marzio, L ;

Cifone, MG .

CRITICAL CARE MEDICINE, 1999, 27 (11) :2413-2417

[6]

DESWAL A, 1997, CIRCULATION S1, V96, P1

[7] ANTICYTOKINE STRATEGIES IN THE TREATMENT OF THE SYSTEMIC INFLAMMATORY RESPONSE SYNDROME [J].

DINARELLO, CA ;

GELFAND, JA ;

WOLFF, SM .

JAMA-JOURNAL OF THE AMERICAN MEDICAL ASSOCIATION, 1993, 269 (14) :1829-1835

[8] INFLUENCE OF AN ANTITUMOR NECROSIS FACTOR MONOCLONAL-ANTIBODY ON CYTOKINE LEVELS IN PATIENTS WITH SEPSIS [J].

FISHER, CJ ;

OPAL, SM ;

DHAINAUT, JF ;

STEPHENS, S ;

ZIMMERMAN, JL ;

NIGHTINGALE, P ;

HARRIS, SJ ;

SCHEIN, RMH ;

PANACEK, EA ;

VINCENT, JL ;

FOULKE, GE ;

WARREN, EL ;

GARRARD, C ;

PARK, G ;

BODMER, MW ;

COHEN, J ;

VANDERLINDEN, C ;

CROSS, AS ;

SADOFF, JC ;

GORECKI, J ;

DUBIN, HG ;

GARNER, C ;

KAYE, W ;

LANORE, JJ ;

MIRA, JP ;

ZIMMERMAN, J ;

DELLINGER, RP ;

TAYLOR, RW ;

DAHL, S ;

SHELLY, M ;

MORTIMER, A ;

EDWARDS, JD ;

KETT, DH ;

QUARTIN, A ;

PENA, MA ;

BAKKER, J ;

ALBERSON, TE ;

WALBY, W ;

RADCLIFFE, J ;

YOUNG, D ;

MCQUILLAM, P ;

BELLINGHAM, G ;

BURMAN, W ;

SADOFF, JS ;

YOUNG, L .

CRITICAL CARE MEDICINE, 1993, 21 (03) :318-327

[9] INHIBITION OF TUMOR-NECROSIS-FACTOR PREVENTS MYOCARDIAL DYSFUNCTION DURING BURN SHOCK [J].

GIROIR, BP ;

HORTON, JW ;

WHITE, DJ ;

MCINTYRE, KL ;

LIN, CQ .

AMERICAN JOURNAL OF PHYSIOLOGY, 1994, 267 (01) :H118-H124

[10] THE TISSUE DISTRIBUTION OF TUMOR-NECROSIS-FACTOR BIOSYNTHESIS DURING ENDOTOXEMIA [J].

GIROIR, BP ;

JOHNSON, JH ;

BROWN, T ;

ALLEN, GL ;

BEUTLER, B .

JOURNAL OF CLINICAL INVESTIGATION, 1992, 90 (03) :693-698

← 1 2 3 →