Heat acclimation alters nitric oxide response in the splanchnic circulation

被引：14

作者：

Haddad, W

Horowitz, M

机构：

[1] Hebrew Univ Jerusalem, Div Physiol, Hadassah Sch Dent Med, IL-91120 Jerusalem, Israel

[2] Hebrew Univ Jerusalem, Div Physiol, Hadassah Med Sch, IL-91120 Jerusalem, Israel

来源：

JOURNAL OF THERMAL BIOLOGY | 1999年 / 24卷 / 5-6期

关键词：

D O I：

10.1016/S0306-4565(99)00054-6

中图分类号：

Q [生物科学];

学科分类号：

07 ; 0710 ; 09 ;

摘要：

The role of nitric oxide (NO) mediated vasorelaxation in splanchnic blood flow during heat stress was studied in rats before and after heat acclimation (1 month, at 34 degrees C). Superior mesenteric artery and portal vein blood hows were measured on-line in the conscious rats during heat stress at 40 degrees C and 42 degrees C before and after LNNA administration. NO mediated vasorelaxation in these vessels was studies in vitro, in response to pilocarpine, Ca ionophore and Na nitroprusside stimulation. The data suggest that NO is an integral part of the thermoregulatory splanchnic vasomotor response, and that heat acclimation enhances its regulatory importance. (C) 2000 Elsevier Science Ltd. All rights reserved.

引用

页码：403 / 408

页数：6

共 19 条

[1]

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[2]

Horowitz M, 1998, NEWS PHYSIOL SCI, V13, P218

[3] Evidence for contribution of effector organ cellular responses to the biphasic dynamics of heat acclimation [J].