Distinct regulation of IRS proteins in adipose tissue from obese aged and dexamethasone-treated rats

被引:23
作者
Caperuto, Luciana C.
Anhe, Gabriel F.
Amanso, Angelica M.
Ribeiro, Luciene M.
Medina, Mayrin C.
Souza, Lilian C.
Carvalho, Olga M. F.
Bordin, Silvana
Saad, Mario J. A.
Carvalho, Carla R. O.
机构
[1] Univ Sao Paulo, Dept Fisiol & Biofis, Inst Ciencias Biomed, BR-05508900 Sao Paulo, Brazil
[2] Univ Estadual Campinas, Fac Ciencias Med, Dept Clin Med, Campinas, SP, Brazil
基金
巴西圣保罗研究基金会;
关键词
adipose tissue; insulin receptor substrates; dexamethasone; aging; insulin resistance;
D O I
10.1385/ENDO:29:3:391
中图分类号
R5 [内科学];
学科分类号
1002 [临床医学]; 100201 [内科学];
摘要
In the present study, we investigated the protein levels and phosphorylation status of the insulin receptor and insulin receptor substrates (IRS-1, IRS-2, and IRS-3) as well as their association with PI(3)-kinase in the rat adipose tissue of two models of insulin resistance: dexamethasone treatment and aging. AKT and atypical PKC phosphorylation detection were also performed. Both models showed decreased insulin-induced IRS-1 and IRS-2 tyrosine phosphorylation, accompanied by reduced protein levels of IRS-1 and IRS-2. Nevertheless, IRS-3 protein level was unchanged in aging but increased in dexamethasone-treated rats. PI(3)-kinase association with IRS-1 was reduced in aged rats, whereas dexamethasone-treated rats showed a reduced IRS-2/ PI(3)-kinase association. However, IRS-3 association with PI(3)-kinase was reduced in both models, as well as insulin-induced AKT and PKC phosphorylation. The alterations described in the present study show that the action of insulin is differently impaired depending on the origin of insulin resistance. These differences might be directly linked to the singular metabolic features of the models we tested.
引用
收藏
页码:391 / 398
页数:8
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