Oncogenic kinases of myeloproliferative disorders induce both protein synthesis and G1 activators

被引：10

作者：

Lelievre, H. ^{[1
]}

Cervera, N.

Finetti, P.

Delhommeau, F.

Vainchenker, W.

Bertucci, F.

Birnbaum, D.

机构：

[1] INSERM, UMR599, Ctr Rech Cancerol Marseille, Oncol Mol Lab, F-13258 Marseille, France

[2] Inst J Paoli I Calmettes, F-13009 Marseille, France

[3] Inst Gustave Roussy, INSERM, U362, F-94805 Villejuif, France

来源：

LEUKEMIA | 2006年 / 20卷 / 10期

关键词：

D O I：

10.1038/sj.leu.2404361

中图分类号：

R73 [肿瘤学];

学科分类号：

100214 ;

摘要：

引用

页码：1885 / 1888

页数：5

共 12 条

[1] The STAT3 transcription factor is a target for the Myc and riboblastoma proteins on the Cdc25A promoter [J].

Barré, B ;

Vigneron, A ;

Coqueret, O .

JOURNAL OF BIOLOGICAL CHEMISTRY, 2005, 280 (16) :15673-15681

[2] Chronic myeloproliferative disorders: a tyrosine kinase tale [J].

De Keersmaecker, K ;

Cools, J .

LEUKEMIA, 2006, 20 (02) :200-205

[3]

Deininger MWN, 2000, CANCER RES, V60, P2049

[4] AKT activity determines sensitivity to mammalian target of rapamycin (mTOR) inhibitors by regulating cyclin D1 and c-myc expression [J].

Gera, JF ;

Mellinghoff, IK ;

Shi, YJ ;

Rettig, MB ;

Tran, C ;

Hsu, JH ;

Sawyers, CL ;

Lichtenstein, AK .

JOURNAL OF BIOLOGICAL CHEMISTRY, 2004, 279 (04) :2737-2746

[5] A unique clonal JAK2 mutation leading to constitutive signalling causes polycythaemia vera [J].

James, C ;

Ugo, V ;

Le Couédic, JP ;

Staerk, J ;

Delhommeau, F ;

Lacout, C ;

Garçon, L ;

Raslova, H ;

Berger, R ;

Bennaceur-Griscelli, A ;

Villeval, JL ;

Constantinescu, SN ;

Casadevall, N ;

Vainchenker, W .

NATURE, 2005, 434 (7037) :1144-1148

[6]

Jena N, 2002, CANCER RES, V62, P535

[7]

LAMOND A, 2005, NUCLEOLAR PROTEOME D

[8] Growth suppression by acute promyelocytic leukemia-associated protein PLZF is mediated by repression of c-myc expression [J].

McConnell, MJ ;

Chevallier, N ;

Berkofsky-Fessler, W ;

Giltnane, JM ;

Malani, RB ;

Staudt, LM ;

Licht, JD .

MOLECULAR AND CELLULAR BIOLOGY, 2003, 23 (24) :9375-9388

[9] BCR-ABL and interleukin 3 promote haematopoietic cell proliferation and survival through modulation of cyclin D2 and p27Kip1 expression [J].

Parada, Y ;

Banerji, L ;

Glassford, J ;

Lea, NC ;

Collado, M ;

Rivas, C ;

Lewis, JL ;

Gordon, MY ;

Thomas, NSB ;

Lam, EWF .

JOURNAL OF BIOLOGICAL CHEMISTRY, 2001, 276 (26) :23572-23580

[10] NoBP, a nuclear fibroblast growth factor 3 binding protein, is cell cycle regulated and promotes cell growth [J].

Reimers, K ;

Antoine, M ;

Zapatka, M ;

Blecken, V ;

Dickson, C ;

Kiefer, P .

MOLECULAR AND CELLULAR BIOLOGY, 2001, 21 (15) :4996-5007

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