Minimal residual disease with TEL-AML1 fusion transcript in childhood acute lymphoblastic leukaemia with t(12;21)

被引:26
作者
Satake, N
Kobayashi, H
Tsunematsu, Y
Kawasaki, H
Horikoshi, Y
Koizumi, S
Kaneko, Y
机构
[1] SAITAMA CANC CTR HOSP,DEPT CANC CHEMOTHERAPY,INA,SAITAMA 362,JAPAN
[2] SAITAMA CANC CTR HOSP,CLIN DEPT 3,HAEMATOL CLIN,INA,SAITAMA 362,JAPAN
[3] NATL CHILDRENS HOSP,SECT HAEMATOL ONCOL,SETAGAYA KU,TOKYO 154,JAPAN
[4] MIE UNIV,FAC MED,DEPT PAEDIAT,TSU,MIE 514,JAPAN
[5] SHIZUOKA CHILDRENS HOSP,SECT HAEMATOL ONCOL,SHIZUOKA,JAPAN
[6] KANAZAWA UNIV,SCH MED,DEPT PAEDIAT,KANAZAWA,ISHIKAWA 920,JAPAN
关键词
minimal residual disease; TEL-AML1 fusion transcript; childhood acute lymphoblastic leukaemia; 12; 21; translocation; RT-PCR; AML1; GENE; LEUKEMIA;
D O I
10.1046/j.1365-2141.1997.762712.x
中图分类号
R5 [内科学];
学科分类号
1002 ; 100201 ;
摘要
We analysed the TEL-AML1 transcript using reverse transcription-polymerase chain reaction (RT-PCR) in order to detect minimal residual disease (MRD) in seven children with t(12;21)-associated B-lineage ALL. Leukaemic cells with the TEL-AML1 transcript appear to be very sensitive to chemotherapy, and may be eradicated in most patients if adequate chemotherapy is given. However, a small number of patients with t(12;21) ALL may relapse under the currently used chemotherapy, and we believe that RT-PCR for detecting MRD with the transcript is a suitable tool for monitoring the efficacy of chemotherapy or impending relapse in these patients. We analysed the TEL-AML1 transcript using reverse transcription-polymerase chain reaction (RT-PCR) in order to detect minimal residual disease (MRD) in seven children with t(12;21)-associated B-lineage ALL. Two sets of primers, TEL exon 5 and AML1 exon 3 or 4, detected two types of transcript in four patients and two other types in two other patients. The two different translocation breakpoints in the AML1 gene with or without splicing out of AML1 exon 3 seemed to result in these four types of transcript in leukaemia samples.
引用
收藏
页码:607 / 611
页数:5
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