Mesenchymal-epithelial interactions in lung development and repair: are modeling and remodeling the same process?

被引:95
作者
Demayo, F
Minoo, P
Plopper, CG
Schuger, L
Shannon, J
Torday, JS [1 ]
机构
[1] Harbor Univ Calif Los Angeles, Res & Educ Inst, Dept Pediat & Obstet & Gynecol, Torrance, CA 90502 USA
[2] Baylor Coll Med, Dept Mol & Cell Biol, Houston, TX 77030 USA
[3] Univ So Calif, Sch Med, Dept Pediat, Div Neonatol,Womens & Childrens Hosp, Los Angeles, CA 90033 USA
[4] Univ Calif Davis, Sch Vet Med, Dept Anat Physiol & Cell Biol, Davis, CA 95616 USA
[5] Wayne State Univ, Sch Med, Dept Pathol, Detroit, MI 48201 USA
[6] Childrens Hosp, Med Ctr, Div Pulm Biol, Cincinnati, OH 45229 USA
关键词
bronchopulmonary dyplasia; smooth muscle; terminal sac;
D O I
10.1152/ajplung.00144.2002
中图分类号
Q4 [生理学];
学科分类号
071003 ;
摘要
We propose that lung morphogenesis and repair are characterized by complex cell-cell interactions of endodermal and mesodermal origin, leading to (or returning back to) an alveolar structure that can effectively exchange gases between the circulation and the alveolar space. We provide the developmental basis for cell/molecular control of lung development and disease, what is known about growth and transcription factors in normal and abnormal lung development, and how endodermal and mesodermal cell origins interact during lung development and disease. The global mechanisms that mediate mesenchymal-epithelial interactions and the plasticity of mesenchymal cells in normal lung development and remodeling provide a functional genomic model that may bring these concepts closer together. We present a synopsis followed by a vertical integration of the developmental and injury/repair mechanisms.
引用
收藏
页码:L510 / L517
页数:8
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