GAD antigen and its significance in Type I diabetes

被引：5

作者：

Hawa, MI ^{[1
]}

Leslie, RDG ^{[1
]}

机构：

[1] St Bartholomews Hosp, Dept Endocrinol, London, England

来源：

JOURNAL OF ENDOCRINOLOGICAL INVESTIGATION | 2002年 / 25卷 / 07期

关键词：

D O I：

10.1007/BF03345077

中图分类号：

R5 [内科学];

学科分类号：

1002 ; 100201 ;

摘要：

引用

页码：576 / 576

页数：1

共 5 条

[1] Identification of naturally processed T cell epitopes from glutamic acid decarboxylase presented in the context of HLA-DR alleles by T lymphocytes of recent onset IDDM patients [J].

Endl, J ;

Otto, H ;

Jung, G ;

Dreisbusch, B ;

Donie, F ;

Stahl, P ;

Elbracht, R ;

Schmitz, G ;

Meinl, E ;

Hummel, M ;

Ziegler, AG ;

Wank, R ;

Schendel, DJ .

JOURNAL OF CLINICAL INVESTIGATION, 1997, 99 (10) :2405-2415

[2] Value of antibodies to islet protein tyrosine phosphatase-like molecule in predicting type 1 diabetes [J].

Hawa, M ;

Rowe, R ;

Lan, MS ;

Notkins, AL ;

Pozzilli, P ;

Christie, MR ;

Leslie, RDG .

DIABETES, 1997, 46 (08) :1270-1275

[3] Antoantigens IA-2 and GAD in type I (insulin-dependent) diabetes [J].

Leslie, RDG ;

Atkinson, MA ;

Notkins, AL .

DIABETOLOGIA, 1999, 42 (01) :3-14

[4] Immune reactivity to glutamic acid decarboxylase 65 in stiff-man syndrome and type 1 diabetes mellitus [J].

Lohmann, T ;

Hawa, M ;

Leslie, RDG ;

Lane, R ;

Picard, J ;

Londei, M .

LANCET, 2000, 356 (9223) :31-35

[5] Modulating autoimmune responses to GAD inhibits disease progression and prolongs islet graft survival in diabetes-prone mice [J].

Tian, JD ;

ClareSalzler, M ;

Herschenfeld, A ;

Middleton, B ;

Newman, D ;

Mueller, R ;

Arita, S ;

Evans, C ;

Atkinson, MA ;

Mullen, Y ;

Sarvetnick, N ;

Tobin, AJ ;

Lehmann, PV ;

Kaufman, DL .

NATURE MEDICINE, 1996, 2 (12) :1348-1353

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