Cerebral arteriolar pathology in a 32-year-old patient with CADASIL

被引：10

作者：

Miao, Q. ^{[1
]}

Kalimo, H.

Bogdanovic, N.

Kostulas, K.

Borjesson-Hanson, A.

Viitanen, M.

机构：

[1] Turku Univ Hosp, Dept Pathol, FIN-20520 Turku, Finland

[2] Univ Turku, Turku, Finland

[3] Karolinska Inst, Neurotec Dept, Stockholm, Sweden

[4] Karolinska Univ Hosp, Stockholm, Sweden

[5] Univ Gothenburg, Sahlgrenska Acad, Sect Psychiat, Gothenburg, Sweden

[6] Univ Uppsala, Dept Pathol, Uppsala, Sweden

[7] Acad Hosp, Uppsala, Sweden

[8] Univ Helsinki, Dept Pathol, Helsinki, Finland

[9] Univ Helsinki Hosp, Helsinki, Finland

[10] Univ Turku, Dept Geriatr Med, Turku, Finland

[11] Turku City Hosp, Turku, Finland

来源：

NEUROPATHOLOGY AND APPLIED NEUROBIOLOGY | 2006年 / 32卷 / 04期

关键词：

D O I：

10.1111/j.1365-2990.2006.00746.x

中图分类号：

R74 [神经病学与精神病学];

学科分类号：

摘要：

[No abstract available]

引用

页码：455 / 458

页数：4

共 11 条

[1] Insidious cognitive decline in CADASIL [J].

Amberla, K ;

Wälijas, M ;

Tuominen, S ;

Almkvist, O ;

Pöyhönen, M ;

Tuisku, S ;

Kalimo, H ;

Viitanen, M .

STROKE, 2004, 35 (07) :1598-1602

[2] Large cerebral artery involvement in CADASIL [J].

Choi, EJ ;

Choi, CG ;

Kim, JS .

NEUROLOGY, 2005, 65 (08) :1322-1324

[3] Scanning laser Doppler flowmetry shows reduced retinal capillary blood flow in CADASIL [J].

Harju, M ;

Tuominen, S ;

Summanen, P ;

Viitanen, M ;

Pöyhönen, M ;

Nikoskelainen, E ;

Kalimo, H ;

Kivelä, T .

STROKE, 2004, 35 (11) :2449-2452

[4] Notch3 mutations in CADASIL, a hereditary adult-onset condition causing stroke and dementia [J].

Joutel, A ;

Corpechot, C ;

Ducros, A ;

Vahedi, K ;

Chabriat, H ;

Mouton, P ;

Alamowitch, S ;

Domenga, V ;

Cecillion, M ;

Marechal, E ;

Maciazek, J ;

Vayssiere, C ;

Cruaud, C ;

Cabanis, EA ;

Ruchoux, MM ;

Weissenbach, J ;

Bach, JF ;

Bousser, MG ;

TournierLasserve, E .

NATURE, 1996, 383 (6602) :707-710

[5]

Kalimo H, 2002, BRAIN PATHOL, V12, P371

[6] Clinicopathological and genetic studies of two further Italian families with cerebral autosomal dominant arteriopathy [J].

Malandrini, A ;

Carrera, P ;

Palmeri, S ;

Cavallaro, T ;

Fabrizi, GM ;

Villanova, M ;

Fattapposta, M ;

Vismara, L ;

Brancolini, V ;

Tanganelli, P ;

Cali, A ;

Morocutti, C ;

Zeviani, M ;

Ferrari, M ;

Guazzi, GC .

ACTA NEUROPATHOLOGICA, 1996, 92 (02) :115-122

[7]

Miao Q, 2004, BRAIN PATHOL, V14, P358

[8] Long-term prognosis and causes of death in CADASIL: a retrospective study in 411 patients [J].

Opherk, C ;

Peters, N ;

Herzog, J ;

Luedtke, R ;

Dichgans, M .

BRAIN, 2004, 127 :2533-2539

[9] Genetic, clinical and pathological studies of CADASIL in Japan: a partial contribution of Notch3 mutations and implications of smooth muscle cell degeneration for the pathogenesis [J].

Santa, Y ;

Uyama, E ;

Chui, DH ;

Arima, M ;

Kotorii, S ;

Takahashi, K ;

Tabira, T .

JOURNAL OF THE NEUROLOGICAL SCIENCES, 2003, 212 (1-2) :79-84

[10] Positron emission tomography examination of cerebral blood flow and glucose metabolism in young CADASIL patients [J].

Tuominen, S ;

Miao, Q ;

Kurki, T ;

Tuisku, S ;

Pöyhönen, M ;

Kalimo, H ;

Viitanen, M ;

Sipilä, HT ;

Bergman, J ;

Rinne, JO .

STROKE, 2004, 35 (05) :1063-1067

← 1 2 →