Host-pathogen interactions:: Subversion and utilization of the NF-κB pathway during infection

被引:149
作者
Tato, CM [1 ]
Hunter, CA [1 ]
机构
[1] Univ Penn, Sch Vet Med, Dept Pathobiol, Philadelphia, PA 19104 USA
关键词
D O I
10.1128/IAI.70.7.3311-3317.2002
中图分类号
R392 [医学免疫学]; Q939.91 [免疫学];
学科分类号
100102 ;
摘要
引用
收藏
页码:3311 / 3317
页数:7
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共 96 条
[1]   The human immunodeficiency virus type 1 accessory protein Vpu induces apoptosis by suppressing the nuclear factor κB-dependent expression of antiapoptotic factors [J].
Akari, H ;
Bour, S ;
Kao, S ;
Adachi, A ;
Strebel, K .
JOURNAL OF EXPERIMENTAL MEDICINE, 2001, 194 (09) :1299-1311
[2]  
Al-Harthi L, 1998, INT J MOL MED, V1, P875
[3]   A SOLUBLE RECEPTOR FOR INTERLEUKIN-1-BETA ENCODED BY VACCINIA VIRUS - A NOVEL MECHANISM OF VIRUS MODULATION OF THE HOST RESPONSE TO INFECTION [J].
ALCAMI, A ;
SMITH, GL .
CELL, 1992, 71 (01) :153-167
[4]   Genetic approaches to study Rel/NF-kappa B/I kappa B function in mice [J].
Attar, RM ;
Caamano, J ;
Carrasco, D ;
Iotsova, V ;
Ishikawa, H ;
Ryseck, RP ;
Weih, F ;
Bravo, R .
SEMINARS IN CANCER BIOLOGY, 1997, 8 (02) :93-101
[5]  
Aune TM, 1999, J IMMUNOL, V162, P5805
[6]   The human immunodeficiency virus type 1 Vpu protein inhibits NF-κB activation by interfering with βTrCP-mediated degradation of IκB [J].
Bour, S ;
Perrin, C ;
Akari, H ;
Strebel, K .
JOURNAL OF BIOLOGICAL CHEMISTRY, 2001, 276 (19) :15920-15928
[7]   A46R and A52R from vaccinia virus are antagonists of host IL-1 and toll-like receptor signaling [J].
Bowie, A ;
Kiss-Toth, E ;
Symons, JA ;
Smith, GL ;
Dower, SK ;
O'Neill, LAJ .
PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA, 2000, 97 (18) :10162-10167
[8]   Toxoplasma gondii tachyzoites inhibit proinflammatory cytokine induction in infected macrophages by preventing nuclear translocation of the transcription factor NF-κB [J].
Butcher, BA ;
Kim, L ;
Johnson, PF ;
Denkers, EY .
JOURNAL OF IMMUNOLOGY, 2001, 167 (04) :2193-2201
[9]  
Caamaño J, 1999, J IMMUNOL, V163, P4453
[10]   NF-κB inhibition causes spontaneous apoptosis in Epstein-Barr virus-transformed lymphoblastoid cells [J].
Cahir-McFarland, ED ;
Davidson, DM ;
Schauer, SL ;
Duong, J ;
Kieff, E .
PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA, 2000, 97 (11) :6055-6060