Amyloid-β:: A vascular sealant that protects against hemorrhage?

被引:38
作者
Atwood, CS [1 ]
Bishop, GM [1 ]
Perry, G [1 ]
Smith, MA [1 ]
机构
[1] Case Western Reserve Univ, Inst Pathol, Cleveland, OH 44106 USA
关键词
D O I
10.1002/jnr.10388
中图分类号
Q189 [神经科学];
学科分类号
071006 ;
摘要
引用
收藏
页码:356 / 356
页数:1
相关论文
共 7 条
[1]   Dramatic aggregation of Alzheimer Aβ by Cu(II) is induced by conditions representing physiological acidosis [J].
Atwood, CS ;
Moir, RD ;
Huang, XD ;
Scarpa, RC ;
Bacarra, NME ;
Romano, DM ;
Hartshorn, MK ;
Tanzi, RE ;
Bush, AI .
JOURNAL OF BIOLOGICAL CHEMISTRY, 1998, 273 (21) :12817-12826
[2]   Neuroinflammatory responses in the Alzheimer's disease brain promote the oxidative post-translational modification of amyloid deposits [J].
Atwood, CS ;
Huang, XD ;
Moir, RD ;
Smith, MA ;
Tanzi, RE ;
Roher, AE ;
Bush, AI ;
Perry, G .
ALZHEIMER'S DISEASE: ADVANCES IN ETIOLOGY, PATHOGENESIS AND THERAPEUTICS, 2001, :341-361
[3]  
Chan CW, 1999, ALZHEIMERS REP, V2, P113
[4]   Amyloid-β junkies [J].
Perry, G ;
Nunomura, A ;
Raina, AK ;
Smith, MA .
LANCET, 2000, 355 (9205) :757-757
[5]   Ischemic rats as a model in the study of the neurobiological role of human β-amyloid peptide.: Time-dependent disappearing diffuse amyloid plaques in brain [J].
Pluta, R ;
Barcikowska, M ;
Misicka, A ;
Lipkowski, AW ;
Spisacka, S ;
Januszewski, S .
NEUROREPORT, 1999, 10 (17) :3615-3619
[6]   BETA-AMYLOID PROTEIN DEPOSITION IN THE BRAIN AFTER SEVERE HEAD-INJURY - IMPLICATIONS FOR THE PATHOGENESIS OF ALZHEIMERS-DISEASE [J].
ROBERTS, GW ;
GENTLEMAN, SM ;
LYNCH, A ;
MURRAY, L ;
LANDON, M ;
GRAHAM, DI .
JOURNAL OF NEUROLOGY NEUROSURGERY AND PSYCHIATRY, 1994, 57 (04) :419-425
[7]  
Smith MA, 2000, ANN NY ACAD SCI, V924, P35