Therapeutic neonatal hepatic gene therapy in mucopolysaccharidosis VII dogs

被引:133
作者
Ponder, KP
Melniczek, JR
Xu, LF
Weil, MA
O'Malley, TM
O'Donnell, PA
Knox, VW
Aguirre, GD
Mazrier, H
Ellinwood, NM
Sleeper, M
Maguire, AM
Volk, SW
Mango, RL
Zweigle, J
Wolfe, JH
Haskins, ME
机构
[1] Univ Penn, Sch Vet Med, Dept Pathobiol, Philadelphia, PA 19104 USA
[2] Univ Penn, Sch Vet Med, Dept Clin Studies, Philadelphia, PA 19104 USA
[3] Washington Univ, Sch Med, Dept Internal Med, St Louis, MO 63110 USA
[4] Washington Univ, Sch Med, Dept Biochem & Mol Biophys, St Louis, MO 63110 USA
[5] Cornell Univ, Coll Vet Med, Baker Inst, Ithaca, NY 14853 USA
[6] Univ Penn, Scheie Eye Inst, Philadelphia, PA 19104 USA
关键词
D O I
10.1073/pnas.192353499
中图分类号
O [数理科学和化学]; P [天文学、地球科学]; Q [生物科学]; N [自然科学总论];
学科分类号
07 ; 0710 ; 09 ;
摘要
Dogs with mucopolysaccharidosis VII (MPS VII) were injected intravenously at 2-3 days of age with a retroviral vector (RV) expressing canine beta-glucuronidase (cGUSB). Five animals received RV alone, and two dogs received hepatocyte growth factor (HGF) before RV in an attempt to increase transduction efficiency. Transduced hepatocytes expanded clonally during normal liver growth and secreted enzyme with mannose 6-phosphate. Serum GUSB activity was stable for up to 14 months at normal levels for the RV-treated dogs, and for 17 months at 67-fold normal for the HGF/RV-treated dog. GUSB activity in other organs was 1.5-60% of normal at 6 months for two RV-treated dogs, which was likely because of uptake of enzyme from blood by the mannose 6-phosphate receptor. The body weights of untreated MPS VII dogs are 50% of normal at 6 months. MPS VII dogs cannot walk or stand after 6 months, and progressively develop eye and heart disease. RV- and HGF/RV-treated MPS VII dogs achieved 87% and 84% of normal body weight, respectively. Treated animals could run at all times of evaluation for 6-17 months because of improvements in bone and joint abnormalities, and had little or no corneal clouding and no mitral valve thickening. Despite higher GUSB expression, the clinical improvements in the HGF/RV-treated dog were similar to those in the RV-treated animals. This is the first successful application of gene therapy in preventing the clinical manifestations of a lysosomal storage disease in a large animal.
引用
收藏
页码:13102 / 13107
页数:6
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