Genomic instability, mutators, and the development of cancer: Is there a role for p53?

[1] A 2-STAGE THEORY OF CARCINOGENESIS IN RELATION TO THE AGE DISTRIBUTION OF HUMAN CANCER [J]. BRITISH JOURNAL OF CANCER, 1957, 11 (02) : 161 - 169

[2] P53 BINDS SINGLE-STRANDED-DNA ENDS AND CATALYZES DNA RENATURATION AND STRAND TRANSFER [J]. PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA, 1994, 91 (01) : 413 - 417

[3] A GENETIC MODEL FOR COLORECTAL TUMORIGENESIS [J]. CELL, 1990, 61 (05) : 759 - 767

[4] THE HUMAN MUTATOR GENE HOMOLOG MSH2 AND ITS ASSOCIATION WITH HEREDITARY NONPOLYPOSIS COLON-CANCER [J]. CELL, 1993, 75 (05) : 1027 - 1038

[5] FISHEL R, 1994, CELL, V77, P167

[6] MULTIPLE-MUTATION THEORY OF CARCINOGENESIS [J]. NATURE, 1958, 181 (4609) : 651 - 652

[7] FISHER JC, 1951, CANCER, V4, P916, DOI 10.1002/1097-0142(195109)4:5<916::AID-CNCR2820040504>3.0.CO

[8] 2-7

[9] DEFECTS IN A CELL-CYCLE CHECKPOINT MAY BE RESPONSIBLE FOR THE GENOMIC INSTABILITY OF CANCER-CELLS [J]. CELL, 1992, 71 (04) : 543 - 546

[10] p53 tumor suppressor gene status and the degree of genomic instability in sporadic colorectal cancers [J]. JOURNAL OF THE NATIONAL CANCER INSTITUTE, 1996, 88 (22) : 1665 - 1670

[1] A 2-STAGE THEORY OF CARCINOGENESIS IN RELATION TO THE AGE DISTRIBUTION OF HUMAN CANCER [J]. BRITISH JOURNAL OF CANCER, 1957, 11 (02) : 161 - 169

[2] P53 BINDS SINGLE-STRANDED-DNA ENDS AND CATALYZES DNA RENATURATION AND STRAND TRANSFER [J]. PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA, 1994, 91 (01) : 413 - 417

[3] A GENETIC MODEL FOR COLORECTAL TUMORIGENESIS [J]. CELL, 1990, 61 (05) : 759 - 767

[4] THE HUMAN MUTATOR GENE HOMOLOG MSH2 AND ITS ASSOCIATION WITH HEREDITARY NONPOLYPOSIS COLON-CANCER [J]. CELL, 1993, 75 (05) : 1027 - 1038

[5] FISHEL R, 1994, CELL, V77, P167

[6] MULTIPLE-MUTATION THEORY OF CARCINOGENESIS [J]. NATURE, 1958, 181 (4609) : 651 - 652

[7] FISHER JC, 1951, CANCER, V4, P916, DOI 10.1002/1097-0142(195109)4:5<916::AID-CNCR2820040504>3.0.CO

[8] 2-7

[9] DEFECTS IN A CELL-CYCLE CHECKPOINT MAY BE RESPONSIBLE FOR THE GENOMIC INSTABILITY OF CANCER-CELLS [J]. CELL, 1992, 71 (04) : 543 - 546

[10] p53 tumor suppressor gene status and the degree of genomic instability in sporadic colorectal cancers [J]. JOURNAL OF THE NATIONAL CANCER INSTITUTE, 1996, 88 (22) : 1665 - 1670