The emerging role of CTLA-4 as an immune attenuator

被引：523

作者：

Thompson, CB ^{[1
]}

Allison, JP ^{[1
]}

机构：

[1] UNIV CALIF BERKELEY,HOWARD HUGHES MED INST,DEPT MOL & CELL BIOL,DIV IMMUNOL,CANC RES LAB,BERKELEY,CA 94720

来源：

IMMUNITY | 1997年 / 7卷 / 04期

关键词：

D O I：

10.1016/S1074-7613(00)80366-0

中图分类号：

R392 [医学免疫学]; Q939.91 [免疫学];

学科分类号：

100102 ;

摘要：

[No abstract available]

引用

页码：445 / 450

页数：6

共 40 条

[1] Die and let live: Eliminating dangerous lymphocytes [J].

Abbas, AK .

CELL, 1996, 84 (05) :655-657

[2]

Alegre ML, 1996, J IMMUNOL, V157, P4762

[3] B7-1 and B7-2 have overlapping, critical roles in immunoglobulin class switching and germinal center formation [J].

Borriello, F ;

Sethna, MP ;

Boyd, SD ;

Schweitzer, AN ;

Tivol, EA ;

Jacoby, D ;

Strom, TB ;

Simpson, EM ;

Freeman, GJ ;

Sharpe, AH .

IMMUNITY, 1997, 6 (03) :303-313

[4] The role of CTLA-4 in the regulation and initiation of T-cell responses [J].

Chambers, CA ;

Krummel, MF ;

Boitel, B ;

Hurwitz, A ;

Sullivan, TJ ;

Fournier, S ;

Cassell, D ;

Brunner, M ;

Allison, JP .

IMMUNOLOGICAL REVIEWS, 1996, 153 :27-46

[5]

CHAMBERS CA, 1997, IN PRESS P NATL ACAD

[6]

Chuang E, 1997, J IMMUNOL, V159, P144

[7] CTLA4 alanine-17 confers genetic susceptibility to Graves' disease and to type 1 diabetes mellitus [J].

Donner, H ;

Rau, H ;

Walfish, PG ;

Braun, J ;

Siegmund, T ;

Finke, R ;

Herwig, J ;

Usadel, KH ;

Badenhoop, K .

JOURNAL OF CLINICAL ENDOCRINOLOGY & METABOLISM, 1997, 82 (01) :143-146

[8] ABSENCE OF B7-DEPENDENT RESPONSES IN CD28-DEFICIENT MICE [J].

GREEN, JM ;

NOEL, PJ ;

SPERLING, AI ;

WALUNAS, TL ;

GRAY, GS ;

BLUESTONE, JA ;

THOMPSON, CB .

IMMUNITY, 1994, 1 (06) :501-508

[9] CTLA4 MEDIATES ANTIGEN-SPECIFIC APOPTOSIS OF HUMAN T-CELLS [J].

GRIBBEN, JG ;

FREEMAN, GJ ;

BOUSSIOTIS, VA ;

RENNERT, P ;

JELLIS, CL ;

GREENFIELD, E ;

BARBER, M ;

RESTIVO, VA ;

KE, XY ;

GRAY, GS ;

NADLER, LM .

PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA, 1995, 92 (03) :811-815

[10] Specific blockade of CTLA-4/B7 interactions results in exacerbated clinical and histologic disease in an actively-induced model of experimental allergic encephalomyelitis [J].

Hurwitz, AA ;

Sullivan, TJ ;

Krummel, MF ;

Sobel, RA ;

Allison, JP .

JOURNAL OF NEUROIMMUNOLOGY, 1997, 73 (1-2) :57-62

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