Viral inhibition of apoptosis

被引：44

作者：

Gillet, G ^{[1
]}

Brun, G ^{[1
]}

机构：

[1] ENS LYON,UMR CNRS 49,LAB BIOL MOL & CELLULAIRE,F-69364 LYON 07,FRANCE

来源：

TRENDS IN MICROBIOLOGY | 1996年 / 4卷 / 08期

关键词：

D O I：

10.1016/0966-842X(96)10047-0

中图分类号：

Q5 [生物化学]; Q7 [分子生物学];

学科分类号：

071010 ; 081704 ;

摘要：

Viral propagation is limited both by the host immune response and by apoptosis of infected cells. Viruses circumvent apoptosis by different mechanisms: direct inhibition of particular proteases involved in cell death, stimulation of anti-death pathways or regulation of the activity of transcription factors monitoring cell survival.

引用

页码：312 / 317

页数：6

共 50 条

[1] AN APOPTOSIS-INHIBITING GENE FROM A NUCLEAR POLYHEDROSIS-VIRUS ENCODING A POLYPEPTIDE WITH CYS/HIS SEQUENCE MOTIF
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[2] ADENOVIRUS-E1B 19-KDA AND BCL-2 PROTEINS INTERACT WITH A COMMON SET OF CELLULAR PROTEINS
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[3] GROWTH-FACTOR MODULATION OF P53-MEDIATED GROWTH ARREST VERSUS APOPTOSIS
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[5] A CONSERVED DOMAIN IN BAK, DISTINCT FROM BH1 AND BH2, MEDIATES CELL-DEATH AND PROTEIN-BINDING FUNCTIONS
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[6] CONTROL OF PROGRAMMED CELL-DEATH BY THE BACULOVIRUS GENES P35 AND IAP
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[J]. MOLECULAR AND CELLULAR BIOLOGY, 1994, 14 (08) : 5212 - 5222
[7] CLEVELAND JL, 1994, ONCOGENE, V9, P2217
[8] AN APOPTOSIS-INHIBITING BACULOVIRUS GENE WITH A ZINC FINGER-LIKE MOTIF
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[J]. JOURNAL OF VIROLOGY, 1993, 67 (04) : 2168 - 2174
[9] ACTIVATION OF THE APOPTOTIC PROTEASE CPP32 BY CYTOTOXIC T-CELL-DERIVED GRANZYME-B
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[10] WILD-TYPE P53 MEDIATES APOPTOSIS BY E1A, WHICH IS INHIBITED BY E1B
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[J]. GENES & DEVELOPMENT, 1993, 7 (04) : 546 - 554

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