A genetic predisposition to hypertension?

被引：7

作者：

Geller, DS ^{[1
]}

机构：

[1] Yale Univ, Sch Med, Nephrol Sect, New Haven, CT USA

来源：

HYPERTENSION | 2004年 / 44卷 / 01期

关键词：

D O I：

10.1161/01.HYP.0000134531.28796.32

中图分类号：

R6 [外科学];

学科分类号：

1002 ; 100210 ;

摘要：

引用

页码：27 / 28

页数：2

共 15 条

[1] Assessing the impact of population stratification on genetic association studies [J].

Freedman, ML ;

Reich, D ;

Penney, KL ;

McDonald, GJ ;

Mignault, AA ;

Patterson, N ;

Gabriel, SB ;

Topol, EJ ;

Smoller, JW ;

Pato, CN ;

Pato, MT ;

Petryshen, TYL ;

Kolonel, LN ;

Lander, ES ;

Sklar, P ;

Henderson, B ;

Hirschhorn, JN ;

Altshuler, D .

NATURE GENETICS, 2004, 36 (04) :388-393

[2] Genome-wide linkage reveals a locus for human essential (primary) hypertension on chromosome 12p [J].

Gong, ML ;

Zhang, HY ;

Schulz, H ;

Lee, YA ;

Sun, K ;

Bähring, S ;

Luft, FC ;

Nürnberg, P ;

Reis, A ;

Rohde, K ;

Ganten, D ;

Hui, RT ;

Hübner, N .

HUMAN MOLECULAR GENETICS, 2003, 12 (11) :1273-1277

[3] BLOOD-PRESSURE CONTROL - SPECIAL ROLE OF THE KIDNEYS AND BODY-FLUIDS [J].

GUYTON, AC .

SCIENCE, 1991, 252 (5014) :1813-1816

[4] Activating mutation of the renal epithelial chloride channel ClC-Kb predisposing to hypertension [J].

Jeck, N ;

Waldegger, S ;

Lampert, A ;

Boehmer, C ;

Waldegger, P ;

Lang, PA ;

Wissinger, B ;

Friedrich, B ;

Risler, T ;

Moehle, R ;

Lang, UE ;

Zill, P ;

Bondy, B ;

Schaeffeler, E ;

Asante-Poku, S ;

Seyberth, H ;

Schwab, M ;

Lang, F .

HYPERTENSION, 2004, 43 (06) :1175-1181

[5] A common sequence variation of the CLCNKB gene strongly activates ClC-Kb chloride channel activity [J].

Jeck, N ;

Waldegger, P ;

Doroszewicz, J ;

Seyberth, H ;

Waldegger, S .

KIDNEY INTERNATIONAL, 2004, 65 (01) :190-197

[6] Evidence for a gene influencing blood pressure on chromosome 17 - Genome scan linkage results for longitudinal blood pressure phenotypes in subjects from the Framingham Heart Study [J].

Levy, D ;

DeStefano, AL ;

Larson, MG ;

O'Donnell, CJ ;

Lifton, RP ;

Gavras, H ;

Cupples, LA ;

Myers, RH .

HYPERTENSION, 2000, 36 (04) :477-483

[7] Molecular mechanisms of human hypertension [J].

Lifton, RP ;

Gharavi, AG ;

Geller, DS .

CELL, 2001, 104 (04) :545-556

[8] Meta-analysis of genetic association studies supports a contribution of common variants to susceptibility to common disease [J].

Lohmueller, KE ;

Pearce, CL ;

Pike, M ;

Lander, ES ;

Hirschhorn, JN .

NATURE GENETICS, 2003, 33 (02) :177-182

[9] Present status of genetic mechanisms in hypertension [J].

Luft, FC .

MEDICAL CLINICS OF NORTH AMERICA, 2004, 88 (01) :1-+

[10] Multilocus linkage of familial hyperkalaemia and hypertension, pseudohypoaldosteronism type II, to chromosomes 1q31-42 and 17p11-q21 [J].

Mansfield, TA ;

Simon, DB ;

Farfel, Z ;

Bia, M ;

Tucci, JR ;

Lebel, M ;

Gutkin, M ;

Vialettes, B ;

Christofilis, MA ;

KauppinenMakelin, R ;

Mayan, H ;

Risch, N ;

Lifton, RP .

NATURE GENETICS, 1997, 16 (02) :202-205

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