Genetics and new approaches to cancer therapy - Commentary

被引：26

作者：

Cavenee, WK ^{[1
]}

机构：

[1] Univ Calif San Diego, Ctr Mol Genet, Dept Med, Ludwig Inst Canc Res, La Jolla, CA 92093 USA

[2] Univ Calif San Diego, Ctr Canc, La Jolla, CA 92093 USA

来源：

CARCINOGENESIS | 2002年 / 23卷 / 05期

关键词：

D O I：

10.1093/carcin/23.5.683

中图分类号：

R73 [肿瘤学];

学科分类号：

100214 ;

摘要：

[No abstract available]

引用

页码：683 / 686

页数：4

共 34 条

[1]

BATRA SK, 1995, CELL GROWTH DIFFER, V6, P1251

[2]

CADENA DL, 1994, J BIOL CHEM, V269, P260

[3] Receptor dimerization is not a factor in the signalling activity of a transforming variant epidermal growth factor receptor (EGFRvIII) [J].

Chu, CT ;

Everiss, KD ;

Wikstrand, CJ ;

Batra, SK ;

Kung, HJ ;

Bigner, DD .

BIOCHEMICAL JOURNAL, 1997, 324 :855-861

[4]

DEPALAZZO IEG, 1993, CANCER RES, V53, P3217

[5] Expression of activated epidermal growth factor receptors, Ras-guanosine triphosphate, and mitogen-activated protein kinase in human glioblastoma multiforme specimens [J].

Feldkamp, MM ;

Lala, P ;

Lau, N ;

Roncari, L ;

Guha, A .

NEUROSURGERY, 1999, 45 (06) :1442-1453

[6]

Han YC, 1996, CANCER RES, V56, P3859

[7] SPECIFIC TARGETING OF A MUTANT, ACTIVATED EGF RECEPTOR FOUND IN GLIOBLASTOMA USING A MONOCLONAL-ANTIBODY [J].

HILLS, D ;

ROWLINSONBUSZA, G ;

GULLICK, WJ .

INTERNATIONAL JOURNAL OF CANCER, 1995, 63 (04) :537-543

[8] The enhanced tumorigenic activity of a mutant epidermal growth factor receptor common in human cancers is mediated by threshold levels of constitutive tyrosine phosphorylation and unattenuated signaling [J].

Huang, HJS ;

Nagane, M ;

Klingbeil, CK ;

Lin, H ;

Nishikawa, R ;

Ji, XD ;

Huang, CM ;

Gill, GN ;

Wiley, HS ;

Cavenee, WK .

JOURNAL OF BIOLOGICAL CHEMISTRY, 1997, 272 (05) :2927-2935

[9] PTB DOMAIN BINDING TO SIGNALING PROTEINS THROUGH A SEQUENCE MOTIF CONTAINING PHOSPHOTYROSINE [J].

KAVANAUGH, WM ;

TURCK, CW ;

WILLIAMS, LT .

SCIENCE, 1995, 268 (5214) :1177-1179

[10]

Luwor RB, 2001, CANCER RES, V61, P5355

← 1 2 3 4 →