New paradigms in oncological therapeutics: redefining combination chemotherapy

被引:4
作者
Brenner, DE [1 ]
机构
[1] Univ Michigan, Med Ctr, Canc Ctr 2150, Ann Arbor, MI 48109 USA
[2] Univ Michigan, Med Ctr, Geriatr Ctr, Ann Arbor, MI USA
[3] VA Med Ctr, Ann Arbor, MI USA
关键词
D O I
10.1093/annonc/mdf349
中图分类号
R73 [肿瘤学];
学科分类号
100214 ;
摘要
引用
收藏
页码:1697 / 1698
页数:2
相关论文
共 13 条
[1]  
Belotti D, 1996, CLIN CANCER RES, V2, P1843
[2]   Multinational study of the efficacy and safety of humanized anti-HER2 monoclonal antibody in women who have HER2-overexpressing metastatic breast cancer that has progressed after chemotherapy for metastatic disease [J].
Cobleigh, MA ;
Vogel, CL ;
Tripathy, D ;
Robert, NJ ;
Scholl, S ;
Fehrenbacher, L ;
Wolter, JM ;
Paton, V ;
Shak, S ;
Lieberman, G ;
Slamon, DJ .
JOURNAL OF CLINICAL ONCOLOGY, 1999, 17 (09) :2639-2648
[3]   Efficacy and safety of imatinib mesylate in advanced gastrointestinal stromal tumors [J].
Demetri, GD ;
von Mehren, M ;
Blanke, CD ;
Van den Abbeele, AD ;
Eisenberg, B ;
Roberts, PJ ;
Heinrich, MC ;
Tuveson, DA ;
Singer, S ;
Janicek, M ;
Fletcher, JA ;
Silverman, SG ;
Silberman, SL ;
Capdeville, R ;
Kiese, B ;
Peng, B ;
Dimitrijevic, S ;
Druker, BJ ;
Corless, C ;
Fletcher, CDM ;
Joensuu, H .
NEW ENGLAND JOURNAL OF MEDICINE, 2002, 347 (07) :472-480
[4]   Perspectives on the development of a molecularly targeted agent [J].
Druker, BJ .
CANCER CELL, 2002, 1 (01) :31-36
[5]   ST1571 (Gleevec™) as a paradigm for cancer therapy [J].
Druker, BJ .
TRENDS IN MOLECULAR MEDICINE, 2002, 8 (04) :S14-S18
[6]   Clinical resistance to STI-571 cancer therapy caused by BCR-ABL gene mutation or amplification [J].
Gorre, ME ;
Mohammed, M ;
Ellwood, K ;
Hsu, N ;
Paquette, R ;
Rao, PN ;
Sawyers, CL .
SCIENCE, 2001, 293 (5531) :876-880
[7]  
Hochhaus A, 2001, SCIENCE, V293, P2163
[8]   APOPTOSIS AND THE PROGNOSTIC-SIGNIFICANCE OF P53 MUTATION [J].
LOWE, SW ;
BODIS, S ;
BARDEESY, N ;
MCCLATCHEY, A ;
REMINGTON, L ;
RULEY, HE ;
FISHER, DE ;
JACKS, T ;
PELLETIER, J ;
HOUSMAN, DE .
COLD SPRING HARBOR SYMPOSIA ON QUANTITATIVE BIOLOGY, 1994, 59 :419-426
[9]   P53-DEPENDENT APOPTOSIS MODULATES THE CYTOTOXICITY OF ANTICANCER AGENTS [J].
LOWE, SW ;
RULEY, HE ;
JACKS, T ;
HOUSMAN, DE .
CELL, 1993, 74 (06) :957-967
[10]   TYROSINE KINASE-ACTIVITY AND TRANSFORMATION POTENCY OF BCR-ABL ONCOGENE PRODUCTS [J].
LUGO, TG ;
PENDERGAST, AM ;
MULLER, AJ ;
WITTE, ON .
SCIENCE, 1990, 247 (4946) :1079-1082