Transglutaminase, gluten and celiac disease: Food for thought

被引:33
作者
Marsh, MN
机构
[1] Hope Hospital,University Department of Medicine
关键词
D O I
10.1038/nm0797-725
中图分类号
Q5 [生物化学]; Q7 [分子生物学];
学科分类号
071010 ; 081704 ;
摘要
Tansglutaminase is identified as the autoantigen or celiac disease (pages 797–801).
引用
收藏
页码:725 / 726
页数:2
相关论文
共 11 条
[1]   Identification of tissue transglutaminase as the autoantigen of celiac disease [J].
Dieterich, W ;
Ehnis, T ;
Bauer, M ;
Donner, P ;
Volta, U ;
Riecken, EO ;
Schuppan, D .
NATURE MEDICINE, 1997, 3 (07) :797-801
[2]  
FERGUSON A, 1987, IMMUNOPATHOLOGY SMAL, P225
[3]   ENDOMYSIAL ANTIBODIES IN THE DIAGNOSIS OF CELIAC-DISEASE AND THE EFFECT OF GLUTEN ON ANTIBODY-TITERS [J].
KUMAR, V ;
LERNER, A ;
VALESKI, JE ;
BEUTNER, EH ;
CHORZELSKI, TP ;
ROSSI, T .
IMMUNOLOGICAL INVESTIGATIONS, 1989, 18 (1-4) :533-544
[4]  
MacDonald TT, 1992, COELIAC DISEASE, P283
[5]  
Marsh M. N., 1992, Coeliac Disease, P136
[6]   GLUTEN, MAJOR HISTOCOMPATIBILITY COMPLEX, AND THE SMALL-INTESTINE - A MOLECULAR AND IMMUNOBIOLOGICAL APPROACH TO THE SPECTRUM OF GLUTEN SENSITIVITY (CELIAC SPRUE) [J].
MARSH, MN .
GASTROENTEROLOGY, 1992, 102 (01) :330-354
[7]  
MARSH MN, J GASTROEN HEPATOL, V8, P270
[8]   GLUTEN SPECIFIC, HLA-DQ RESTRICTED T-CELLS FROM CELIAC MUCOSA PRODUCE CYTOKINES WITH THL OR THO PROFILE DOMINATED BY INTERFERON-GAMMA [J].
NILSEN, EM ;
LUNDIN, KEA ;
KRAJCI, P ;
SCOTT, H ;
SOLLID, LM ;
BRANDTZAEG, P .
GUT, 1995, 37 (06) :766-776
[9]   Proteolytic degradation of intestinal mucosal extracellular matrix after lamina propria T cell activation [J].
Pender, SLF ;
Lionetti, P ;
Murch, SH ;
Wathan, N ;
MacDonald, TT .
GUT, 1996, 39 (02) :284-290
[10]   CELIAC SYNDROME - BIOCHEMICAL-MECHANISMS AND THE MISSING PEPTIDASE HYPOTHESIS REVISTED [J].
PETERS, TJ ;
BJARNASON, I .
GUT, 1984, 25 (09) :913-918