Resolving the nitric oxide paradox in acute tissue damage

被引：24

作者：

Billiar, TR ^{[1
]}

Harbrecht, BG ^{[1
]}

机构：

[1] PRESBYTERIAN UNIV HOSP, DEPT SURG, PITTSBURGH, PA 15213 USA

来源：

GASTROENTEROLOGY | 1997年 / 113卷 / 04期

关键词：

D O I：

10.1053/gast.1997.v113.agast971131405

中图分类号：

R57 [消化系及腹部疾病];

学科分类号：

摘要：

[No abstract available]

引用

页码：1405 / 1407

页数：3

共 13 条

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[4] NITRIC-OXIDE AND PROSTAGLANDINS INTERACT TO PREVENT HEPATIC DAMAGE DURING MURINE ENDOTOXEMIA
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[5] INHIBITION OF NITRIC-OXIDE SYNTHESIS DURING ENDOTOXEMIA PROMOTES INTRAHEPATIC THROMBOSIS AND AN OXYGEN RADICAL-MEDIATED HEPATIC-INJURY
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[6] INHIBITION OF NITRIC-OXIDE SYNTHASE DURING HEMORRHAGIC-SHOCK INCREASES HEPATIC-INJURY
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[7] NITROGEN OXIDE-INDUCED AUTOPROTECTION IN ISOLATED RAT HEPATOCYTES
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[8] OU J, IN PRESS NITRIC OXID
[9] Targeting nitric oxide (NO) delivery in vivo. Design of a liver-selective NO donor prodrug that blocks tumor necrosis factor-alpha-induced apoptosis and toxicity in the liver
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[10] NITRIC-OXIDE DILATES TIGHT JUNCTIONS AND DEPLETES ATP IN CULTURED CACO-2BBE INTESTINAL EPITHELIAL MONOLAYERS
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[J]. AMERICAN JOURNAL OF PHYSIOLOGY-GASTROINTESTINAL AND LIVER PHYSIOLOGY, 1995, 268 (02): : G361 - G373

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