Mechanisms underlying abnormal trafficking and expansion of malignant progenitors in CML: BCR/ABL-induced defects in integrin function in CML

被引:43
作者
Salesse, S
Verfaillie, CM
机构
[1] Univ Minnesota, Ctr Canc, Stem Cell Inst, Minneapolis, MN 55455 USA
[2] Univ Minnesota, Ctr Canc, Dept Med, Minneapolis, MN 55455 USA
关键词
CML; BCR/ABL; integrin; adhesion; proliferation; molecular mechanisms;
D O I
10.1038/sj.onc.1206088
中图分类号
Q5 [生物化学]; Q7 [分子生物学];
学科分类号
071010 ; 081704 ;
摘要
[No abstract available]
引用
收藏
页码:8605 / 8611
页数:7
相关论文
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