First-generation adenovirus vectors shorten survival time in a murine model of sepsis

被引:28
作者
Doerschug, K
Sanlioglu, S
Flaherty, DM
Wilson, RL
Yarovinsky, T
Monick, MM
Engelhardt, JF
Hunninghake, GW
机构
[1] Univ Iowa, Coll Med, Dept Internal Med, Div Pulm Crit Care & Occupat Med, Iowa City, IA 52242 USA
[2] Univ Iowa, Coll Med, Vet Adm Med Ctr, Iowa City, IA 52242 USA
[3] Univ Iowa, Coll Med, Ctr Gene Therapy, Iowa City, IA 52242 USA
[4] Univ Iowa, Coll Med, Dept Anat & Cell Biol, Iowa City, IA 52242 USA
[5] Akdeniz Univ, Coll Med, Dept Med Biol & Genet, Antalya, Turkey
关键词
D O I
10.4049/jimmunol.169.11.6539
中图分类号
R392 [医学免疫学]; Q939.91 [免疫学];
学科分类号
100102 ;
摘要
Adverse immunological reactions to adenoviral vectors have significantly impacted the utility of this virus for treating genetic and environmentally induced diseases. In this study, we evaluate the effect of adenoviral vectors on an animal model of sepsis. Systemic delivery of first-generation adenoviral vectors to septic mice (cecal ligation and puncture) resulted in a shortened survival time. This effect was not observed with second-generation or inactivated first-generation vectors. The accelerated death was accompanied by a number of important changes in the disease. These changes included increased liver cell apoptosis (including Kupffer cells) and a marked increase in liver bacterial load. In the lung, the combination induced an increase in bacterial load, as well as greater lung injury. In the serum, the combination was associated with decreased TNF-alpha levels and an increase in bacterial load. Finally, a profound degree of lymphocyte apoptosis was observed in these animals. These observations suggest that prior exposure to first-generation adenovirus gene therapy vectors may worsen the outcome of some forms of sepsis.
引用
收藏
页码:6539 / 6545
页数:7
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