Activation-associated mitochondrial hyperpolarization hijacks T cells toward an apoptosis-sensitized phenotype

被引：15

作者：

Matarrese, P

Cauda, R

Malorni, I

机构：

[1] Ist Super Sanita, Dept Ultrastruct, I-00161 Rome, Italy

[2] Univ Cattolica Sacro Cuore, Dept Infect Dis, Rome, Italy

来源：

CELL DEATH AND DIFFERENTIATION | 2003年 / 10卷 / 05期

关键词：

D O I：

10.1038/sj.cdd.4401212

中图分类号：

Q5 [生物化学]; Q7 [分子生物学];

学科分类号：

071010 ; 081704 ;

摘要：

引用

页码：609 / 611

页数：3

共 26 条

[1]

Banki K, 1999, J IMMUNOL, V162, P1466

[2]

Bedner E, 1999, CYTOMETRY, V35, P181, DOI 10.1002/(SICI)1097-0320(19990301)35:3<181::AID-CYTO1>3.0.CO

[3]

2-5

[4] Mitochondrial cytochrome c release in apoptosis occurs upstream of DEVD-specific caspase activation and independently of mitochondrial transmembrane depolarization [J].

Bossy-Wetzel, E ;

Newmeyer, DD ;

Green, DR .

EMBO JOURNAL, 1998, 17 (01) :37-49

[5]

Capobianchi MR, 1996, J BIOL REG HOMEOS AG, V10, P83

[6] PROTECTIVE EFFECT OF N-ACETYLCYSTEINE IN TUMOR NECROSIS FACTOR-ALPHA-INDUCED APOPTOSIS IN U937 CELLS - THE ROLE OF MITOCHONDRIA [J].

COSSARIZZA, A ;

FRANCESCHI, C ;

MONTI, D ;

SALVIOLI, S ;

BELLESIA, E ;

RIVABENE, R ;

BIONDO, L ;

RAINALDI, G ;

TINARI, A ;

MALORNI, W .

EXPERIMENTAL CELL RESEARCH, 1995, 220 (01) :232-240

[7] Cell type specific involvement of death receptor and mitochondrial pathways in drug-induced apoptosis [J].

Fulda, S ;

Meyer, E ;

Friesen, C ;

Susin, SA ;

Kroemer, G ;

Debatin, KM .

ONCOGENE, 2001, 20 (09) :1063-1075

[8]

Gergely P, 2002, ARTHRITIS RHEUM-US, V46, P175, DOI 10.1002/1529-0131(200201)46:1<175::AID-ART10015>3.0.CO

[9]

2-H

[10] Mitochondria hyperpolarization is an early event in oxidized low-density lipoprotein-induced apoptosis in Caco-2 intestinal cells [J].

Giovannini, C ;

Matarrese, P ;

Scazzocchio, B ;

Sanchez, M ;

Masella, R ;

Malorni, W .

FEBS LETTERS, 2002, 523 (1-3) :200-206

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