Myb proteins in life, death and differentiation

被引:180
作者
Weston, K [1 ]
机构
[1] Inst Canc Res, Chester Beatty Labs, CRC, Ctr Cell & Mol Biol, London SW3 6JB, England
关键词
D O I
10.1016/S0959-437X(98)80065-8
中图分类号
Q2 [细胞生物学];
学科分类号
071009 ; 090102 ;
摘要
Myb transcription factors are crucial to the control of proliferation and differentiation in a number of cell types but their mechanism of action is unclear. Regulation of Myb proteins by phosphorylation and intermolecular cooperation has recently been demonstrated, together with a new role for the proteins, in the control of apoptosis.
引用
收藏
页码:76 / 81
页数:6
相关论文
共 69 条
[1]   MODULATION OF C-MYB-INDUCED TRANSCRIPTION ACTIVATION BY A PHOSPHORYLATION SITE NEAR THE NEGATIVE REGULATORY DOMAIN [J].
AZIZ, N ;
MIGLARESE, MR ;
HENDRICKSON, RC ;
SHABANOWITZ, J ;
STURGILL, TW ;
HUNT, DF ;
BENDER, TP .
PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA, 1995, 92 (14) :6429-6433
[2]   DIFFERENTIAL EXPRESSION OF C-MYB MESSENGER-RNA IN MURINE-B LYMPHOMAS BY A BLOCK TO TRANSCRIPTION ELONGATION [J].
BENDER, TP ;
THOMPSON, CB ;
KUEHL, WM .
SCIENCE, 1987, 237 (4821) :1473-1476
[3]  
Bennett JD, 1996, ONCOGENE, V13, P1073
[4]  
BIES J, 1995, CANCER RES, V55, P501
[5]   Oncogenic activation of c-Myb by carboxyl-terminal truncation leads to decreased proteolysis by the ubiquitin-26S proteasome pathway [J].
Bies, J ;
Wolff, L .
ONCOGENE, 1997, 14 (02) :203-212
[6]  
Blau J, 1996, MOL CELL BIOL, V16, P2044
[7]   c-Myb trans-activates the human DNA topoisomerase II alpha gene promoter [J].
Brandt, TL ;
Fraser, DJ ;
Leal, S ;
Halandras, PM ;
Kroll, AR ;
Kroll, DJ .
JOURNAL OF BIOLOGICAL CHEMISTRY, 1997, 272 (10) :6278-6284
[8]   Core binding factor cannot synergistically activate the myeloperoxidase proximal enhancer in immature myeloid cells without c-Myb [J].
BritosBray, M ;
Friedman, AD .
MOLECULAR AND CELLULAR BIOLOGY, 1997, 17 (09) :5127-5135
[9]   THE ANTIPROLIFERATIVE ACTIVITY OF C-MYB AND C-MYC ANTISENSE OLIGONUCLEOTIDES IN SMOOTH-MUSCLE CELLS IS CAUSED BY A NONANTISENSE MECHANISM [J].
BURGESS, TL ;
FISHER, EF ;
ROSS, SL ;
BREADY, JV ;
QIAN, YX ;
BAYEWITCH, LA ;
COHEN, AM ;
HERRERA, CJ ;
HU, SSF ;
KRAMER, TB ;
LOTT, FD ;
MARTIN, FH ;
PIERCE, GF ;
SIMONET, L ;
FARRELL, CL .
PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA, 1995, 92 (09) :4051-4055
[10]   ESTROGEN-DEPENDENT ALTERATIONS IN DIFFERENTIATION STATE OF MYELOID CELLS CAUSED BY A V-MYB ESTROGEN-RECEPTOR FUSION PROTEIN [J].
BURK, O ;
KLEMPNAUER, KH .
EMBO JOURNAL, 1991, 10 (12) :3713-3719