Deoxycholate-induced colitis is markedly attenuated in Nos2 knockout mice in association with modulation of gene expression profiles

被引:22
作者
Bernstein, Harris [1 ]
Holubec, Hana
Bernstein, Carol
Ignatenko, Natalia A.
Gerner, Eugene
Dvorak, Katerina
Besselsen, David
Blohm-Mangone, Karen Ann
Padilla-Torres, Jose
Dvorakova, Barbora
Garewal, Harinder
Payne, Claire M.
机构
[1] Univ Arizona, Coll Med, Dept Cell Biol & Anat, Tucson, AZ 85724 USA
[2] Univ Arizona, Coll Med, Dept Internal Med, Tucson, AZ 85724 USA
[3] Univ Arizona, Ctr Canc, Tucson, AZ 85724 USA
[4] Univ Arizona, Univ Anim Care, Tucson, AZ 85724 USA
关键词
colitis; nitric oxide synthase; deoxycholate; inflammatory bowel disease; colon cancer;
D O I
10.1007/s10620-006-9608-0
中图分类号
R57 [消化系及腹部疾病];
学科分类号
摘要
Nos2 knockout mice were compared to wild-type mice for susceptibility to colitis in response to a diet supplemented with deoxycholate, a bile acid increased in the colon of individuals on a high-fat diet. Wild-type mice fed a fat-related diet, supplemented with 0.2% DOC, develop colonic inflammation associated with increases in nitrosative stress, proliferation, oxidative DNA/RNA damage, and angiogenesis, as well as altered expression of numerous genes. However, Nos2 knockout mice fed a diet supplemented with deoxycholate were resistant to these alterations. In particular, 35 genes were identified whose expression was significantly altered at the mRNA level in deoxycholate-fed Nos2(+/+) mice but not in deoxycholate-fed Nos2(-/-) mice. Some of these alterations in NOS2-dependent gene expression correspond to those reported in human inflammatory bowel disease. Overall, our results indicate that NOS2 expression is necessary for the development of deoxycholate-induced colitis in mice, a unique dietary-related model of colitis.
引用
收藏
页码:628 / 642
页数:15
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