Molecular regulation of pancreatic β-cell mass development, maintenance, and expansion

被引:207
作者
Ackermann, Amanda M.
Gannon, Maureen
机构
[1] Vanderbilt Univ, Med Ctr, Dept Med, Nashville, TN 37232 USA
[2] Vanderbilt Univ, Med Ctr, Dept Mol Physiol & Biophys, Nashville, TN 37232 USA
[3] Vanderbilt Univ, Med Ctr, Program Dev Biol, Nashville, TN 37232 USA
关键词
D O I
10.1677/JME-06-0053
中图分类号
R5 [内科学];
学科分类号
1002 ; 100201 ;
摘要
Pancreatic beta-cells are responsible for producing all of the insulin required by an organism to maintain glucose homeostasis. Defects in development, maintenance, or expansion of beta-cell mass can result in impaired glucose metabolism and diabetes. Thus, identifying the molecular regulators of these processes may provide new therapeutic targets for diabetes. Additionally, understanding the processes of beta-cell differentiation and proliferation may allow for in vitro cultivation of beta-cells in sufficient amounts to be transplanted into patients with diabetes. This review addresses many of the transcription factors and signaling pathways that play a role in early pancreatic development and endocrine cell (specifically beta-cell) differentiation, conditions that influence beta-cell mass development and molecular regulators of beta-cell proliferation and apoptosis that are responsible for maintaining and expanding beta-cell mass.
引用
收藏
页码:193 / 206
页数:14
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