Mycobacterium tuberculosis and the complement system -: Response

被引:1
作者
Brown, E [1 ]
Schorey, J [1 ]
机构
[1] Washington Univ, Sch Med, Div Infect Dis, Dept Med,Dept Mol Microbiol, St Louis, MO 63110 USA
关键词
D O I
10.1016/S0966-842X(97)01202-X
中图分类号
Q5 [生物化学]; Q7 [分子生物学];
学科分类号
071010 ; 081704 ;
摘要
引用
收藏
页码:49 / 50
页数:2
相关论文
共 9 条
[1]   Disruption of the Cr2 locus results in a reduction in B-1a cells and in an impaired B cell response to T-dependent antigen [J].
Ahearn, JM ;
Fischer, MB ;
Croix, D ;
Goerg, S ;
Ma, MH ;
Xia, JR ;
Zhou, XN ;
Howard, RG ;
Rothstein, TL ;
Carroll, MC .
IMMUNITY, 1996, 4 (03) :251-262
[2]   A novel role for the beta 2 integrin CD11b/CD18 in neutrophil apoptosis: A homeostatic mechanism in inflammation [J].
Coxon, A ;
Rieu, P ;
Barkalow, FJ ;
Askari, S ;
Sharpe, AH ;
vonAndrian, UH ;
Arnaout, MA ;
Mayadas, TN .
IMMUNITY, 1996, 5 (06) :653-666
[3]  
FUJIMURA Y, 1986, Gastroenterologia Japonica, V21, P325
[4]   Abrogation of the alternative complement pathway by targeted deletion of murine factor B [J].
Matsumoto, M ;
Fukuda, W ;
Circolo, A ;
Goellner, J ;
StraussSchoenberger, J ;
Wang, XF ;
Fujita, S ;
Hidvegi, T ;
Chaplin, DD ;
Colten, HR .
PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA, 1997, 94 (16) :8720-8725
[5]   Markedly impaired humoral immune response in mice deficient in complement receptors 1 and 2 [J].
Molina, H ;
Holers, VM ;
Li, B ;
Fang, YF ;
Mariathasan, S ;
Goellner, J ;
StraussSchoenberger, J ;
Karr, RW ;
Chaplin, DD .
PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA, 1996, 93 (08) :3357-3361
[6]   ROLE OF M CELLS AND MACROPHAGES IN THE ENTRANCE OF MYCOBACTERIUM-PARATUBERCULOSIS INTO DOMES OF ILEAL PEYERS PATCHES IN CALVES [J].
MOMOTANI, E ;
WHIPPLE, DL ;
THIERMANN, AB ;
CHEVILLE, NF .
VETERINARY PATHOLOGY, 1988, 25 (02) :131-137
[7]   A macrophage invasion mechanism of pathogenic mycobacteria [J].
Schorey, JS ;
Carroll, MC ;
Brown, EJ .
SCIENCE, 1997, 277 (5329) :1091-1093
[8]   STUDIES OF GROUP-B STREPTOCOCCAL INFECTION IN MICE DEFICIENT IN COMPLEMENT COMPONENT C3 OR C4 DEMONSTRATE AN ESSENTIAL ROLE FOR COMPLEMENT IN BOTH INNATE AND ACQUIRED-IMMUNITY [J].
WESSELS, MR ;
BUTKO, P ;
MA, MH ;
WARREN, HB ;
LAGE, AL ;
CARROLL, MC .
PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA, 1995, 92 (25) :11490-11494
[9]   Cytokine regulation of the macrophage (M phi) system studied using the colony stimulating factor-1-deficient op/op mouse [J].
WiktorJedrzejczak, W ;
Gordon, S .
PHYSIOLOGICAL REVIEWS, 1996, 76 (04) :927-947