TREM-1 ligand expression on platelets enhances neutrophil activation

被引:175
作者
Haselmayer, Philipp
Grosse-Hovest, Ludger
von Landenberg, Philipp
Schild, Hansjoerg
Radsak, Markus P.
机构
[1] Johannes Gutenberg Univ Mainz, Inst Immunol, D-55131 Mainz, Germany
[2] Univ Tubingen, Dept Immunol, Inst Cell Biol, Tubingen, Germany
[3] Johannes Gutenberg Univ Mainz, Univ Hosp, Dept Clin Chem, D-6500 Mainz, Germany
[4] Johannes Gutenberg Univ Mainz, Univ Hosp, Dept Med 3, D-6500 Mainz, Germany
关键词
D O I
10.1182/blood-2007-01-069195
中图分类号
R5 [内科学];
学科分类号
1002 ; 100201 ;
摘要
The triggering receptor expressed on myeloid cells 1 (TREM-1) plays an important role in the innate immune response related to severe infections and sepsis. Modulation of TREM-1-associated activation improves the outcome in rodent models for pneumonia and sepsis. However, the identity and occurrence of the natural TREM-1 ligands are so far unknown, impairing the further understanding of the biology of this receptor. Here, we report the presence of a ligand for TREM-1 on human platelets. Using a recombinant TREM-1 fusion protein, we demonstrate specific binding of TREM-1 to platelets. TREM-1-specific signals are required for the platelet-induced augmentation of polymorphonuclear leukocyte (PMN) effector functions (provoked by LPS). However, TREM-1 interaction with its ligand is not required for platelet/PMN complex formation, which is dependent on integrins and selectins. Taken together, the results indicate that the TREM-1 ligand is expressed by platelets, and the TREM-1/ligand interaction contributes to the amplification of LPS-induced PMN activation. Our results shed new light on our understanding of TREM-1 and its role in the innate inflammatory response in infections and might contribute to the development of future concepts to treat sepsis.
引用
收藏
页码:1029 / 1035
页数:7
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