The tumor necrosis factor ligand and receptor families

[1] EFFICACY AND SAFETY OF MONOCLONAL-ANTIBODY TO HUMAN TUMOR-NECROSIS-FACTOR-ALPHA IN PATIENTS WITH SEPSIS SYNDROME - A RANDOMIZED, CONTROLLED, DOUBLE-BLIND, MULTICENTER CLINICAL-TRIAL [J]. JAMA-JOURNAL OF THE AMERICAN MEDICAL ASSOCIATION, 1995, 273 (12): : 934 - 941

[2] ABERRANT TRANSCRIPTION CAUSED BY THE INSERTION OF AN EARLY TRANSPOSABLE ELEMENT IN AN INTRON OF THE FAS ANTIGEN GENE OF LPR MICE [J]. PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA, 1993, 90 (05) : 1756 - 1760

[3] AGGARWAL BB, 1984, J BIOL CHEM, V259, P686

[4] CD40 LIGAND GENE DEFECTS RESPONSIBLE FOR X-LINKED HYPER-IGM SYNDROME [J]. SCIENCE, 1993, 259 (5097) : 990 - 993

[5] DIFFERENCES DEFINED BY BONE-MARROW TRANSPLANTATION SUGGEST THAT LPR AND GLD ARE MUTATIONS OF GENES ENCODING AN INTERACTING PAIR OF MOLECULES [J]. JOURNAL OF EXPERIMENTAL MEDICINE, 1990, 172 (05) : 1367 - 1375

[6] PROTECTION AGAINST ENDOTOXIC-SHOCK BY A TUMOR-NECROSIS-FACTOR RECEPTOR IMMUNOADHESIN [J]. PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA, 1991, 88 (23) : 10535 - 10539

[7] CRYSTAL-STRUCTURE OF THE SOLUBLE HUMAN 55 KD TNF RECEPTOR-HUMAN TNF-BETA COMPLEX - IMPLICATIONS FOR TNF RECEPTOR ACTIVATION [J]. CELL, 1993, 73 (03) : 431 - 445

[8] CHIMERIC TUMOR-NECROSIS-FACTOR RECEPTORS WITH CONSTITUTIVE SIGNALING ACTIVITY [J]. PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA, 1995, 92 (12) : 5376 - 5380

[9] PURIFICATION OF CACHECTIN, A LIPOPROTEIN-LIPASE SUPPRESSING HORMONE SECRETED BY ENDOTOXIN-INDUCED RAW 264.7 CELLS [J]. JOURNAL OF EXPERIMENTAL MEDICINE, 1985, 161 (05) : 984 - 995

[10] AN EVOLUTIONARY AND FUNCTIONAL-APPROACH TO THE TNF RECEPTOR/LIGAND FAMILY [J]. MICROBIAL PATHOGENESIS AND IMMUNE RESPONSE, 1994, 730 : 118 - 133

[1] EFFICACY AND SAFETY OF MONOCLONAL-ANTIBODY TO HUMAN TUMOR-NECROSIS-FACTOR-ALPHA IN PATIENTS WITH SEPSIS SYNDROME - A RANDOMIZED, CONTROLLED, DOUBLE-BLIND, MULTICENTER CLINICAL-TRIAL [J]. JAMA-JOURNAL OF THE AMERICAN MEDICAL ASSOCIATION, 1995, 273 (12): : 934 - 941

[2] ABERRANT TRANSCRIPTION CAUSED BY THE INSERTION OF AN EARLY TRANSPOSABLE ELEMENT IN AN INTRON OF THE FAS ANTIGEN GENE OF LPR MICE [J]. PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA, 1993, 90 (05) : 1756 - 1760

[3] AGGARWAL BB, 1984, J BIOL CHEM, V259, P686

[4] CD40 LIGAND GENE DEFECTS RESPONSIBLE FOR X-LINKED HYPER-IGM SYNDROME [J]. SCIENCE, 1993, 259 (5097) : 990 - 993

[5] DIFFERENCES DEFINED BY BONE-MARROW TRANSPLANTATION SUGGEST THAT LPR AND GLD ARE MUTATIONS OF GENES ENCODING AN INTERACTING PAIR OF MOLECULES [J]. JOURNAL OF EXPERIMENTAL MEDICINE, 1990, 172 (05) : 1367 - 1375

[6] PROTECTION AGAINST ENDOTOXIC-SHOCK BY A TUMOR-NECROSIS-FACTOR RECEPTOR IMMUNOADHESIN [J]. PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA, 1991, 88 (23) : 10535 - 10539

[7] CRYSTAL-STRUCTURE OF THE SOLUBLE HUMAN 55 KD TNF RECEPTOR-HUMAN TNF-BETA COMPLEX - IMPLICATIONS FOR TNF RECEPTOR ACTIVATION [J]. CELL, 1993, 73 (03) : 431 - 445

[8] CHIMERIC TUMOR-NECROSIS-FACTOR RECEPTORS WITH CONSTITUTIVE SIGNALING ACTIVITY [J]. PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA, 1995, 92 (12) : 5376 - 5380

[9] PURIFICATION OF CACHECTIN, A LIPOPROTEIN-LIPASE SUPPRESSING HORMONE SECRETED BY ENDOTOXIN-INDUCED RAW 264.7 CELLS [J]. JOURNAL OF EXPERIMENTAL MEDICINE, 1985, 161 (05) : 984 - 995

[10] AN EVOLUTIONARY AND FUNCTIONAL-APPROACH TO THE TNF RECEPTOR/LIGAND FAMILY [J]. MICROBIAL PATHOGENESIS AND IMMUNE RESPONSE, 1994, 730 : 118 - 133