Intermediate filaments and their associated proteins: multiple dynamic personalities

被引：56

作者：

Houseweart, MK

Cleveland, DW

机构：

[1] Univ Calif San Diego, Ludwig Inst Canc Res, La Jolla, CA 92093 USA

[2] Univ Calif San Diego, Div Cellular & Mol Med, La Jolla, CA 92093 USA

[3] Univ Calif San Diego, Dept Med & Neurosci, La Jolla, CA 92093 USA

来源：

CURRENT OPINION IN CELL BIOLOGY | 1998年 / 10卷 / 01期

关键词：

D O I：

10.1016/S0955-0674(98)80091-4

中图分类号：

Q2 [细胞生物学];

学科分类号：

071009 ; 090102 ;

摘要：

A fusion of mouse and human genetics has now proven that intermediate filaments form a flexible scaffold essential for structuring cytoplasm in a variety of cell contexts. In some cases, the formation of this scaffold is achieved through a newly identified family of intermediate-filament-associated proteins that form cross-bridges between intermediate filaments and other cytoskeletal elements, including actin and microtubules.

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页码：93 / 101

页数：9

相关论文

共 60 条

[1] Keratin biochemistry [J].

Albers, KM .

CLINICS IN DERMATOLOGY, 1996, 14 (04) :309-320

[2]

ANDRA K, 1997, IN PRESS GENES DEV

[3] Dystonin expression in the developing nervous system predominates in the neurons that degenerate in dystonia musculorum mutant mice [J].

Bernier, G ;

Brown, A ;

Dalpe, G ;

DeRepentigny, Y ;

Mathieu, M ;

Kothary, R .

MOLECULAR AND CELLULAR NEUROSCIENCE, 1995, 6 (06) :509-520

[4] Cloning and characterization of mouse ACF7, a novel member of the dystonin subfamily of actin binding proteins [J].

Bernier, G ;

Mathieu, M ;

DeRepentigny, Y ;

Vidal, SM ;

Kothary, R .

GENOMICS, 1996, 38 (01) :19-29

[5] THE MOUSE DYSTONIA MUSCULORUM GENE IS A NEURAL ISOFORM OF BULLOUS PEMPHIGOID ANTIGEN-1 [J].

BROWN, A ;

BERNIER, G ;

MATHIEU, M ;

ROSSANT, J ;

KOTHARY, R .

NATURE GENETICS, 1995, 10 (03) :301-306

[6] DEFECTIVE AXONAL-TRANSPORT IN A TRANSGENIC MOUSE MODEL OF AMYOTROPHIC-LATERAL-SCLEROSIS [J].

COLLARD, JF ;

COTE, F ;

JULIEN, JP .

NATURE, 1995, 375 (6526) :61-64

[7] MICE LACKING VIMENTIN DEVELOP AND REPRODUCE WITHOUT AN OBVIOUS PHENOTYPE [J].

COLUCCIGUYON, E ;

PORTIER, MM ;

DUNIA, I ;

PAULIN, D ;

POURNIN, S ;

BABINET, C .

CELL, 1994, 79 (04) :679-694

[8] PROGRESSIVE NEURONOPATHY IN TRANSGENIC MICE EXPRESSING THE HUMAN NEUROFILAMENT HEAVY GENE - A MOUSE MODEL OF AMYOTROPHIC-LATERAL-SCLEROSIS [J].

COTE, F ;

COLLARD, JF ;

JULIEN, JP .

CELL, 1993, 73 (01) :35-46

[9] Developmental expression of BPAG1-n: Insights into the spastic ataxia and gross neurologic degeneration in dystonia musculorum mice [J].

Dowling, J ;

Yang, YM ;

Wollmann, R ;

Reichardt, LF ;

Fuchs, E .

DEVELOPMENTAL BIOLOGY, 1997, 187 (02) :131-142

[10] NEUROFILAMENT-DEFICIENT AXONS AND PERIKARYAL AGGREGATES IN VIABLE TRANSGENIC MICE EXPRESSING A NEUROFILAMENT-BETA-GALACTOSIDASE FUSION PROTEIN [J].

EYER, J ;

PETERSON, A .

NEURON, 1994, 12 (02) :389-405

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