Transgenic models for Alzheimer's disease

被引:7
作者
Duff, K [1 ]
机构
[1] Mayo Clin, Dept Pharmacol, Jacksonville, FL 32224 USA
关键词
D O I
10.1046/j.1365-2990.1998.00006.x
中图分类号
R74 [神经病学与精神病学];
学科分类号
摘要
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页码:101 / 103
页数:3
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共 16 条
[1]   RELEASE OF EXCESS AMYLOID BETA-PROTEIN FROM A MUTANT AMYLOID BETA-PROTEIN PRECURSOR [J].
CAI, XD ;
GOLDE, TE ;
YOUNKIN, SG .
SCIENCE, 1993, 259 (5094) :514-516
[2]   EARLY-ONSET ALZHEIMERS-DISEASE CAUSED BY MUTATIONS AT CODON-717 OF THE BETA-AMYLOID PRECURSOR PROTEIN GENE [J].
CHARTIERHARLIN, MC ;
CRAWFORD, F ;
HOULDEN, H ;
WARREN, A ;
HUGHES, D ;
FIDANI, L ;
GOATE, A ;
ROSSOR, M ;
ROQUES, P ;
HARDY, J ;
MULLAN, M .
NATURE, 1991, 353 (6347) :844-846
[3]   MUTATION OF THE BETA-AMYLOID PRECURSOR PROTEIN IN FAMILIAL ALZHEIMERS-DISEASE INCREASES BETA-PROTEIN PRODUCTION [J].
CITRON, M ;
OLTERSDORF, T ;
HAASS, C ;
MCCONLOGUE, L ;
HUNG, AY ;
SEUBERT, P ;
VIGOPELFREY, C ;
LIEBERBURG, I ;
SELKOE, DJ .
NATURE, 1992, 360 (6405) :672-674
[4]   GENE DOSE OF APOLIPOPROTEIN-E TYPE-4 ALLELE AND THE RISK OF ALZHEIMERS-DISEASE IN LATE-ONSET FAMILIES [J].
CORDER, EH ;
SAUNDERS, AM ;
STRITTMATTER, WJ ;
SCHMECHEL, DE ;
GASKELL, PC ;
SMALL, GW ;
ROSES, AD ;
HAINES, JL ;
PERICAKVANCE, MA .
SCIENCE, 1993, 261 (5123) :921-923
[5]   The presenilin genes: A new gene family involved in Alzheimer disease pathology [J].
Cruts, M ;
Hendriks, L ;
Van Broeckhoven, C .
HUMAN MOLECULAR GENETICS, 1996, 5 :1449-1455
[6]   Increased amyloid-beta 42(43) in brains of mice expressing mutant presenilin 1 [J].
Duff, K ;
Eckman, C ;
Zehr, C ;
Yu, X ;
Prada, CM ;
Pereztur, J ;
Hutton, M ;
Buee, L ;
Harigaya, Y ;
Yager, D ;
Morgan, D ;
Gordon, MN ;
Holcomb, L ;
Refolo, L ;
Zenk, B ;
Hardy, J ;
Younkin, S .
NATURE, 1996, 383 (6602) :710-713
[7]   ALZHEIMER-TYPE NEUROPATHOLOGY IN TRANSGENIC MICE OVEREXPRESSING V717F BETA-AMYLOID PRECURSOR PROTEIN [J].
GAMES, D ;
ADAMS, D ;
ALESSANDRINI, R ;
BARBOUR, R ;
BERTHELETTE, P ;
BLACKWELL, C ;
CARR, T ;
CLEMENS, J ;
DONALDSON, T ;
GILLESPIE, F ;
GUIDO, T ;
HAGOPIAN, S ;
JOHNSONWOOD, K ;
KHAN, K ;
LEE, M ;
LEIBOWITZ, P ;
LIEBERBURG, I ;
LITTLE, S ;
MASLIAH, E ;
MCCONLOGUE, L ;
MONTOYAZAVALA, M ;
MUCKE, L ;
PAGANINI, L ;
PENNIMAN, E ;
POWER, M ;
SCHENK, D ;
SEUBERT, P ;
SNYDER, B ;
SORIANO, F ;
TAN, H ;
VITALE, J ;
WADSWORTH, S ;
WOLOZIN, B ;
ZHAO, J .
NATURE, 1995, 373 (6514) :523-527
[8]   SEGREGATION OF A MISSENSE MUTATION IN THE AMYLOID PRECURSOR PROTEIN GENE WITH FAMILIAL ALZHEIMERS-DISEASE [J].
GOATE, A ;
CHARTIERHARLIN, MC ;
MULLAN, M ;
BROWN, J ;
CRAWFORD, F ;
FIDANI, L ;
GIUFFRA, L ;
HAYNES, A ;
IRVING, N ;
JAMES, L ;
MANT, R ;
NEWTON, P ;
ROOKE, K ;
ROQUES, P ;
TALBOT, C ;
PERICAKVANCE, M ;
ROSES, A ;
WILLIAMSON, R ;
ROSSOR, M ;
OWEN, M ;
HARDY, J .
NATURE, 1991, 349 (6311) :704-706
[9]   TRANSGENIC MOUSE-BRAIN HISTOPATHOLOGY RESEMBLES EARLY ALZHEIMERS-DISEASE [J].
HIGGINS, LS ;
HOLTZMAN, DM ;
RABIN, J ;
MOBLEY, WC ;
CORDELL, B .
ANNALS OF NEUROLOGY, 1994, 35 (05) :598-607
[10]   Correlative memory deficits, A beta elevation, and amyloid plaques in transgenic mice [J].
Hsiao, K ;
Chapman, P ;
Nilsen, S ;
Eckman, C ;
Harigaya, Y ;
Younkin, S ;
Yang, FS ;
Cole, G .
SCIENCE, 1996, 274 (5284) :99-102