Delayed onset massive oedema and deterioration in traumatic brain injury

被引:5
作者
Kohta, Masaaki
Minami, Hiroaki
Tanaka, Kazuhiro
Kuwamura, Keiichi
Kondoh, Takeshi
Kohmura, Eiji
机构
[1] Kobe Univ, Grad Sch Med, Dept Neurosurg, Chuo Ku, Kobe, Hyogo 6500017, Japan
[2] Awaji Hosp, Dept Neurosurg, Sumoto, Japan
关键词
head injury; cerebral vasospasm; brain contusion; delayed deterioration; brain oedema;
D O I
10.1016/j.jocn.2006.01.042
中图分类号
R74 [神经病学与精神病学];
学科分类号
摘要
A 52-year-old man fell from standing and a computed tomography (CT) scan revealed traumatic intracerebral haematoma and subarachnoid haemorrhage in the temporal cortex. He was treated without surgery and discharged. On day 30 after the accident, he had no neurological deficit. On day 37 he complained of headache and urinary incontinence, and on day 39 he was hospitalized due to progressive neurological deterioration (reduced conciousness, dilated pupils, and left herniplegia). A CT scan revealed a diffuse low-density in the right cerebral hemisphere with marked midline shift. Emergency decompressive craniectomy and right temporal lobectomy were performed. Angiography after surgery revealed moderate vasospasm in the right middle and anterior cerebral arteries. The patient remained severely disabled. Delayed onset neurological deterioration can be caused by brain oedema and vasospasm after traumatic brain injury, despite an intervening period of improvement. (c) 2006 Elsevier Ltd. All rights reserved.
引用
收藏
页码:167 / 170
页数:4
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