Viewpoint: The prothrombotic state in heart failure: A maladaptive inflammatory response?

被引：33

作者：

Chong, Aun-Yeong ^{[1
]}

Lip, Gregory Y. H. ^{[1
]}

机构：

[1] Univ Birmingham, Haemostasis Thrombosis & Vasc Biol Unit, Dept Med, City Hosp, Birmingham B18 7QH, W Midlands, England

来源：

EUROPEAN JOURNAL OF HEART FAILURE | 2007年 / 9卷 / 02期

关键词：

prothrombotic state; inflammation; heart failure;

D O I：

10.1016/j.ejheart.2006.05.009

中图分类号：

R5 [内科学];

学科分类号：

1002 ; 100201 ;

摘要：

Patients with heart failure (HF) are at an increased risk of stroke, sudden death and venous thromboembolism, which are all linked to thrombus formation (thrombogenesis). The present 'viewpoint' article will discuss how the prothrombotic state in HF may be perpetuated by a chronic inflammatory state that is maladaptive. Indeed, there is considerable evidence that thrombogenesis and endothelial (dys)function can be intimately linked to inflammation in HF. (c) 2006 European Society of Cardiology. Published by Elsevier B.V. All rights reserved.

引用

收藏

页码：124 / 128

页数：5

相关论文

共 64 条

[1] How to RECOVER from RENAISSANCE? The significance of the results of RECOVER, RENAISSANCE, RENEWAL and ATTACH [J].

Anker, SD ;

Coats, AJS .

INTERNATIONAL JOURNAL OF CARDIOLOGY, 2002, 86 (2-3) :123-130

[2] Cytokine-mediated apoptosis in cardiac myocytes - The role of inducible nitric oxide synthase induction and peroxynitrite generation [J].

Arstall, MA ;

Sawyer, DB ;

Fukazawa, R ;

Kelly, RA .

CIRCULATION RESEARCH, 1999, 85 (09) :829-840

[3] NEURAL NETWORKS FOR POWER-SYSTEMS ALARM HANDLING [J].

BALL, N ;

KIERNAN, L ;

WARWICK, K ;

CAHILL, E ;

ESP, D ;

MACQUEEN, J .

NEUROCOMPUTING, 1992, 4 (1-2) :5-8

[4]

BALLIGAND JL, 1994, J BIOL CHEM, V269, P27580

[5]

BERETZ A, 1990, BIORHEOLOGY, V27, P455

[6] C-Reactive protein in acute myocardial infarction: Association with heart failure [J].

Berton, G ;

Cordiano, R ;

Palmieri, R ;

Pianca, S ;

Pagliara, V ;

Palatini, P .

AMERICAN HEART JOURNAL, 2003, 145 (06) :1094-+

[7] MECHANISM OF THE TUMOR-NECROSIS-FACTOR ALPHA-MEDIATED INDUCTION OF ENDOTHELIAL TISSUE FACTOR [J].

BIERHAUS, A ;

ZHANG, YM ;

DENG, YH ;

MACKMAN, N ;

QUEHENBERGER, P ;

HAASE, M ;

LUTHER, T ;

MULLER, M ;

BOHRER, H ;

GRETEN, J ;

MARTIN, E ;

BAEUERLE, PA ;

WALDHERR, R ;

KISIEL, W ;

ZIEGLER, R ;

STERN, DM ;

NAWROTH, PP .

JOURNAL OF BIOLOGICAL CHEMISTRY, 1995, 270 (44) :26419-26432

[8] Endothelial cells undergoing apoptosis become proadhesive for nonactivated platelets [J].

Bombeli, T ;

Schwartz, BR ;

Harlan, JM .

BLOOD, 1999, 93 (11) :3831-3838

[9] Apoptotic vascular endothelial cells become procoagulant [J].

Bombeli, T ;

Karsan, A ;

Tait, JF ;

Harlan, JM .

BLOOD, 1997, 89 (07) :2429-2442

[10] Cardiac resynchronization and death from progressive heart failure - A meta-analysis of randomized controlled trials [J].

Bradley, DJ ;

Bradley, EA ;

Baughman, KL ;

Berger, RD ;

Calkins, H ;

Goodman, SN ;

Kass, DA ;

Powe, NR .

JAMA-JOURNAL OF THE AMERICAN MEDICAL ASSOCIATION, 2003, 289 (06) :730-740

← 1 2 3 4 5 6 7 →