Action myoclonus-renal failure syndrome: characterization of a unique cerebro-renal disorder

被引:60
作者
Badhwar, A
Berkovic, SF
Dowling, JP
Gonzales, M
Narayanan, S
Brodtmann, A
Berzen, L
Caviness, J
Trenkwalder, C
Winkelmann, J
Rivest, J
Lambert, M
Hernandez-Cossio, O
Carpenter, S
Andermann, F
Andermann, E
机构
[1] Montreal Neurol Hosp & Inst, Neurogenet Unit, Montreal, PQ H3A 2B4, Canada
[2] Montreal Neurol Hosp & Inst, Epilepsy Serv, Montreal, PQ H3A 2B4, Canada
[3] McGill Univ, Dept Human Genet, Montreal, PQ, Canada
[4] McGill Univ, Dept Neurol & Neurosurg, Montreal, PQ, Canada
[5] McGill Univ, Dept Pediat, Montreal, PQ H3A 2T5, Canada
[6] McGill Univ, MRS Unit, McConnell Brain Imaging Ctr, Montreal, PQ H3A 2T5, Canada
[7] Hop St Justine, Dept Med Genet, Montreal, PQ H3T 1C5, Canada
[8] Ctr Hosp Univ Sherbrooke, Serv Neurol, Sherbrooke, PQ, Canada
[9] Univ British Columbia, British Columbia Neuropsychiat Program, Dept Psychiat, Vancouver, BC V5Z 1M9, Canada
[10] Univ Melbourne, Epilepsy Res Ctr, Dept Med, Parkville, Vic 3052, Australia
[11] Alfred Hosp, Dept Pathol, Melbourne, Vic, Australia
[12] Royal Melbourne Hosp, Dept Anat Pathol, Melbourne, Vic, Australia
[13] Royal Melbourne Hosp, Dept Neurol, Melbourne, Vic, Australia
[14] Mayo Clin, Parkinsons Dis & Other Movement Disorders Ctr, Scottsdale, AZ USA
[15] Univ Gottingen, Paracelsus Elena Klin, Kassel, Germany
[16] Max Planck Inst Psychiat, D-80804 Munich, Germany
[17] Natl Res Ctr, Inst Human Genet, Munich, Germany
[18] Hosp NS Pilar, Inst Neurol Pilar, Curitiba, Parana, Brazil
[19] Hosp Sao Joao, Serv Anat Patol, Oporto, Portugal
基金
加拿大健康研究院;
关键词
action myoclonus; renal failure; progressive myoclonus epilepsy; cerebro-renal disorder; autosomal recessive inheritance;
D O I
10.1093/brain/awh263
中图分类号
R74 [神经病学与精神病学];
学科分类号
摘要
Action myoclonus-renal failure syndrome (AMRF) is a distinctive form of progressive myoclonus epilepsy associated with renal dysfunction. The syndrome was not recognized prior to the advent of dialysis and renal transplantation because of its rapidly fatal course if renal failure is untreated. The first and only description of AMRF was in four French Canadian patients in three families (Andermann et al., 1986). We now describe 15 individuals with AMRF from five countries, including a follow-up of the four French Canadian patients, allowing a more complete characterization of this disease. Our 15 patients with AMRF belong to nine different families. Segregation analyses were compatible with autosomal recessive inheritance. In addition, our findings show that AMRF can present with either renal or neurological features. Tremor (onset 17-26 years, mean 19.8 years, median 19 years) and progressively disabling action myoclonus (onset 14-29 years, mean 21.7 years, median 21 years), with infrequent generalized seizures (onset 20-28 years, mean 22.7 years, median 22 years) and cerebellar features are characteristic. Proteinuria, detected between ages 9 and 30 years in all cases, progressed to renal failure in 12 out of 15 patients within 0-8 years after proteinuria detection. Brain autopsy in two patients revealed extraneuronal pigment accumulation. Renal biopsies showed collapsing glomerulopathy, a severe variant of focal glomerulosclerosis. This study extends the AMRF phenotype, and demonstrates a more extensive ethnic and geographic distribution of a syndrome originally believed to be confined to individuals of French Canadian ancestry. The independent progression of neurological and renal disorders in AMRF suggests a unitary molecular lesion with pleiotropic effects. Our results demonstrate that the renal lesion in AMRF is a recessive form of collapsing glomerulopathy. Genes identified for focal segmental glomerulosclerosis and involved with the function of the glomerular basement membrane and related proteins are thus good candidates. Treatment can improve quality of life and extend the lifespan of these patients. Dialysis and renal transplantation are effective for the renal but not the neurological features, which continue to progress even in the presence of normalized renal function; the latter can be managed with anti-myoclonic and anti-epileptic drugs.
引用
收藏
页码:2173 / 2182
页数:10
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