Transfers and transitions: Parent-offspring conflict, genomic imprinting, and the evolution of human life history

被引：73

作者：

Haig, David ^{[1
]}

机构：

[1] Harvard Univ, Dept Organism & Evolutionary Biol, Cambridge, MA 02138 USA

来源：

PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA | 2010年 / 107卷

关键词：

Beckwith-Wiedemann syndrome; genomic imprinting; Prader-Willi syndrome; weaning; PRADER-WILLI-SYNDROME; SILVER-RUSSELL-SYNDROME; MATERNAL UNIPARENTAL DISOMY; BECKWITH-WIEDEMANN-SYNDROME; INTERGENERATIONAL TRANSFERS; HUMAN CHILDHOOD; GROWTH; CHILDREN; AGE; HYPOTHESES;

D O I：

10.1073/pnas.0904111106

中图分类号：

O [数理科学和化学]; P [天文学、地球科学]; Q [生物科学]; N [自然科学总论];

学科分类号：

07 ; 0710 ; 09 ;

摘要：

Human offspring are weaned earlier than the offspring of other great apes but take longer to reach nutritional independence. An analysis of human disorders of imprinted genes suggests genes of paternal origin, expressed in infants, have been selected to favor more intense suckling than genes of maternal origin. The same analysis suggests that genes of maternal origin may favor slower childhood growth but earlier sexual maturation. These observations are consistent with a hypothesis in which slow maturation was an adaptation of offspring that reduced maternal fitness, whereas early weaning was an adaptation of mothers that reduced the fitness of individual offspring.

引用

页码：1731 / 1735

页数：5

共 59 条

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