Viral and host proteins involved in picornavirus life cycle

被引:146
作者
Lin, Jing-Yi [1 ,2 ]
Chen, Tzu-Chun [1 ,2 ]
Weng, Kuo-Feng [1 ,2 ,3 ]
Chang, Shih-Cheng [1 ]
Chen, Li-Lien [1 ,2 ,3 ]
Shih, Shin-Ru [1 ,2 ,3 ,4 ]
机构
[1] Chang Gung Univ, Res Ctr Emerging Viral Infect, Tao Yuan, Taiwan
[2] Chang Gung Univ, Dept Med Biotechnol & Lab Sci, Tao Yuan, Taiwan
[3] Chang Gung Univ, Grad Program Biomed Sci, Tao Yuan, Taiwan
[4] Natl Hlth Res Inst, Div Biotechnol & Pharmaceut Res, Zhunan, Taiwan
关键词
TRACT-BINDING-PROTEIN; RIBOSOME ENTRY SITE; MOUTH-DISEASE VIRUS; HEPATITIS-A VIRUS; ACTING REPLICATION ELEMENT; COXSACKIEVIRUS 2B PROTEIN; DEPENDENT RNA-POLYMERASE; NEGATIVE-STRAND RNA; TRANSLATION INITIATION-FACTOR; POLIOVIRUS-INFECTED CELLS;
D O I
10.1186/1423-0127-16-103
中图分类号
Q2 [细胞生物学];
学科分类号
071009 ; 090102 ;
摘要
Picornaviruses cause several diseases, not only in humans but also in various animal hosts. For instance, human enteroviruses can cause hand-foot-and-mouth disease, herpangina, myocarditis, acute flaccid paralysis, acute hemorrhagic conjunctivitis, severe neurological complications, including brainstem encephalitis, meningitis and poliomyelitis, and even death. The interaction between the virus and the host is important for viral replication, virulence and pathogenicity. This article reviews studies of the functions of viral and host factors that are involved in the life cycle of picornavirus. The interactions of viral capsid proteins with host cell receptors is discussed first, and the mechanisms by which the viral and host cell factors are involved in viral replication, viral translation and the switch from translation to RNA replication are then addressed. Understanding how cellular proteins interact with viral RNA or viral proteins, as well as the roles of each in viral infection, will provide insights for the design of novel antiviral agents based on these interactions.
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页数:14
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