How DNA lesions are turned into mutations within cells?

被引:177
作者
Pagès, V [1 ]
Fuchs, RPP [1 ]
机构
[1] Univ Strasbourg 1, UPR 9003 CNRS, ESBS, UPR Conventionnee, F-67400 Strasbourg, France
关键词
DNA damage tolerance; lesion bypass polymerases; error-prone DNA repair; origin of mutations; mutagens and carcinogens;
D O I
10.1038/sj.onc.1206006
中图分类号
Q5 [生物化学]; Q7 [分子生物学];
学科分类号
071010 ; 081704 ;
摘要
Genomes of all living organisms are constantly injured by endogenous and exogenous agents that modify the chemical integrity of DNA and in turn challenge its informational content. Despite the efficient action of numerous repair systems that remove lesions in DNA in an error-free manner, some lesions, that escape these repair mechanisms, are present when DNA is being replicated. Although replicative DNA polymerases are usually unable to copy past such lesions, it was recently discovered that cells are equipped with specialized DNA polymerases that will assist the replicative polymerase during the process of Translesion Synthesis (TLS). These TLS polymerases exhibit relaxed fidelity that allows them to copy past lesions in DNA with an inherent risk of generating mutations at high frequency. We present recent aspects related to the genetics and biochemistry of TLS and highlight some of the remaining hot topics of this field.
引用
收藏
页码:8957 / 8966
页数:10
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