Transgenic myocardial overexpression of fibroblast growth factor-1 increases coronary artery density and branching

被引:69
作者
Fernandez, B
Buehler, A
Wolfram, S
Kostin, S
Espanion, G
Franz, WM
Niemann, H
Doevendans, PA
Schaper, W
Zimmermann, R
机构
[1] Max Planck Inst, Dept Expt Cardiol, D-61231 Bad Nauheim, Germany
[2] Inst Tierzucht & Tierverhalten, Dept Biotechnol, D-3057 Neustadt, Germany
[3] Med Univ Lubeck, Med Klin 2, Lubeck, Germany
[4] Univ Limburg, Cardiovasc Res Inst, NL-6200 MD Maastricht, Netherlands
[5] Kerckhoff Clin, Bad Nauheim, Germany
关键词
fibroblast growth factor-1; transgenic; hearts; coronary arteries;
D O I
10.1161/01.RES.87.3.207
中图分类号
R5 [内科学];
学科分类号
1002 ; 100201 ;
摘要
Fibroblast growth factor (FGF)-1 plays important roles during myocardial and coronary morphogenesis. FGF-1 is also involved in the physiological response of the adult heart against ischemia, which includes cardiomyocyte protection and vascular growth. In the present study, we have generated transgenic mice with specific myocardial overexpression of the gene. Transgene expression was verified by Northern blot, and increased FGF-1 protein content was assessed by Western blot and immunoconfocal microscopy. Anatomic, histomorphological, and ultrastructural analyses revealed no major morphological or developmental abnormalities of transgenic hearts. Capillary density was unaltered, whereas the density of coronary arteries, especially arterioles, was significantly increased, as was the number of branches of the main coronary arteries. In addition, the coronary flow was significantly enhanced in transgenic mice ex vivo. These differences in the anatomic pattern of the coronary vasculature are established during the second month of postnatal life. The present findings demonstrate an important role of FGF-1 in the differentiation and growth of the coronary system and suggest that it is a key regulatory molecule of the differentiation of the arterial system.
引用
收藏
页码:207 / 213
页数:7
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